Pushpak Bhattacharjee

Kolkata, WB 700054, India

Last Updated: 09/26/2026

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Found 22 publications

Pathogenic Mutations in the C2A Domain of Dysferlin form Amyloid that Activates the Inflammasome.

Pathogenic Mutations in the C2A Domain of Dysferlin form Amyloid that Activates the Inflammasome.

Journal: bioRxiv : the preprint server for biology

Published: May 10, 2023

Retraction Notice to: FoxP3 Acts as a Cotranscription Factor with STAT3 in Tumor-Induced Regulatory T Cells.

Retraction Notice to: FoxP3 Acts as a Cotranscription Factor with STAT3 in Tumor-Induced Regulatory T Cells.

Journal: Immunity

Published: September 15, 2021

The lactate receptor GPR81 promotes breast cancer growth via a paracrine mechanism involving antigen-presenting cells in the tumor microenvironment.

The lactate receptor GPR81 promotes breast cancer growth via a paracrine mechanism involving antigen-presenting cells in the tumor microenvironment.

Journal: Oncogene

Published: June 04, 2019

Andrographolide binds to ATP-binding pocket of VEGFR2 to impede VEGFA-mediated tumor-angiogenesis.

Andrographolide binds to ATP-binding pocket of VEGFR2 to impede VEGFA-mediated tumor-angiogenesis.

Journal: Scientific reports

Published: July 25, 2018

Cholesterol-dependent cytolysins impair pro-inflammatory macrophage responses.

Cholesterol-dependent cytolysins impair pro-inflammatory macrophage responses.

Journal: Scientific reports

Published: December 11, 2017

Providence of the CD25+ KIR+ CD127- FOXP3- CD8+ T-cell subset determines the dynamics of tumor immune surveillance.

Providence of the CD25+ KIR+ CD127- FOXP3- CD8+ T-cell subset determines the dynamics of tumor immune surveillance.

Journal: Immunology and cell biology

Published: August 17, 2017

GPR81, a Cell-Surface Receptor for Lactate, Regulates Intestinal Homeostasis and Protects Mice from Experimental Colitis.

GPR81, a Cell-Surface Receptor for Lactate, Regulates Intestinal Homeostasis and Protects Mice from Experimental Colitis.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: April 26, 2017

WITHDRAWN: Curcumin shifts RAS-induced pro-proliferative MEK/ERK-signaling toward pro-apoptotic p38MAPK/JNK1-signaling, triggering p53 activation and apoptosis.

WITHDRAWN: Curcumin shifts RAS-induced pro-proliferative MEK/ERK-signaling toward pro-apoptotic p38MAPK/JNK1-signaling, triggering p53 activation and apoptosis.

Journal: The Journal of biological chemistry

Published: March 07, 2017

Intrinsic repair protects cells from pore-forming toxins by microvesicle shedding.

Intrinsic repair protects cells from pore-forming toxins by microvesicle shedding.

Journal: Cell death and differentiation

Published: August 29, 2016

Sulphur alters NFκB-p300 cross-talk in favour of p53-p300 to induce apoptosis in non-small cell lung carcinoma.

Sulphur alters NFκB-p300 cross-talk in favour of p53-p300 to induce apoptosis in non-small cell lung carcinoma.

Journal: International journal of oncology

Published: March 25, 2015

Cross-talk between endoplasmic reticulum (ER) stress and the MEK/ERK pathway potentiates apoptosis in human triple negative breast carcinoma cells: role of a dihydropyrimidone, nifetepimine.

Cross-talk between endoplasmic reticulum (ER) stress and the MEK/ERK pathway potentiates apoptosis in human triple negative breast carcinoma cells: role of a dihydropyrimidone, nifetepimine.

Journal: The Journal of biological chemistry

Published: December 21, 2014

Curcumin inhibits breast cancer stem cell migration by amplifying the E-cadherin/β-catenin negative feedback loop.

Curcumin inhibits breast cancer stem cell migration by amplifying the E-cadherin/β-catenin negative feedback loop.

Journal: Stem cell research & therapy

Published: April 12, 2014

Showing 1-12 of 22

Last Updated: 09/26/2026