Qi Shi

Beijing, China

Last Updated: 09/26/2026

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Found 10 publications

Accumulation of Prion Triggers the Enhanced Glycolysis via Activation of AMKP Pathway in Prion-Infected Rodent and Cell Models.

Accumulation of Prion Triggers the Enhanced Glycolysis via Activation of AMKP Pathway in Prion-Infected Rodent and Cell Models.

Journal: Molecular neurobiology

Published: February 19, 2023

Presence of anti-IgLON5 antibody in a case of sporadic Creutzfeldt-Jakob disease with sleep disturbance as a prominent symptom.

Presence of anti-IgLON5 antibody in a case of sporadic Creutzfeldt-Jakob disease with sleep disturbance as a prominent symptom.

Journal: Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology

Published: August 23, 2022

Proteomics profiling for the global and acetylated proteins of papillary thyroid cancers.

Proteomics profiling for the global and acetylated proteins of papillary thyroid cancers.

Journal: Proteome science

Published: December 31, 2021

Genetic Prion Disease: Insight from the Features and Experience of China National Surveillance for Creutzfeldt-Jakob Disease.

Genetic Prion Disease: Insight from the Features and Experience of China National Surveillance for Creutzfeldt-Jakob Disease.

Journal: Neuroscience bulletin

Published: January 29, 2021

T188K-Familial Creutzfeldt-Jacob Disease, Predominant Among Chinese, has a Reactive Pattern in CSF RT-QuIC Different from D178N-Fatal Familial Insomnia and E200K-Familial CJD.

T188K-Familial Creutzfeldt-Jacob Disease, Predominant Among Chinese, has a Reactive Pattern in CSF RT-QuIC Different from D178N-Fatal Familial Insomnia and E200K-Familial CJD.

Journal: Neuroscience bulletin

Published: August 16, 2018

The genetic Creutzfeldt-Jakob disease with E200K mutation: analysis of clinical, genetic and laboratory features of 30 Chinese patients.

The genetic Creutzfeldt-Jakob disease with E200K mutation: analysis of clinical, genetic and laboratory features of 30 Chinese patients.

Journal: Scientific reports

Published: June 29, 2018

Aberrant Alterations of Mitochondrial Factors Drp1 and Opa1 in the Brains of Scrapie Experiment Rodents.

Aberrant Alterations of Mitochondrial Factors Drp1 and Opa1 in the Brains of Scrapie Experiment Rodents.

Journal: Journal of molecular neuroscience : MN

Published: August 29, 2016

Increases of Galectin-1 and its S-nitrosylated form in the Brain Tissues of Scrapie-Infected Rodent Models and Human Prion Diseases.

Increases of Galectin-1 and its S-nitrosylated form in the Brain Tissues of Scrapie-Infected Rodent Models and Human Prion Diseases.

Journal: Molecular neurobiology

Published: January 12, 2016

The Brain NO Levels and NOS Activities Ascended in the Early and Middle Stages and Descended in the Terminal Stage in Scrapie-Infected Animal Models.

The Brain NO Levels and NOS Activities Ascended in the Early and Middle Stages and Descended in the Terminal Stage in Scrapie-Infected Animal Models.

Journal: Molecular neurobiology

Published: November 20, 2015

Human Prion disease with a T188K mutation in Chinese: a case report.

Human Prion disease with a T188K mutation in Chinese: a case report.

Journal: Cases journal

Published: December 11, 2008

Showing 1-10 of 10

Last Updated: 09/26/2026