Qi Yu

Guangzhou, China

Last Updated: 09/26/2026

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Found 13 publications

BMAL1 deficiency exacerbates HDM-induced asthma by promoting JUN-mediated lipid peroxidation and airway epithelial ferroptosis.

BMAL1 deficiency exacerbates HDM-induced asthma by promoting JUN-mediated lipid peroxidation and airway epithelial ferroptosis.

Journal: Redox biology

Published: May 19, 2026

ac4C modification of PDK4 by NAT10 promotes pulmonary fibrosis by reprogramming mitochondrial dynamics in fibroblasts.

ac4C modification of PDK4 by NAT10 promotes pulmonary fibrosis by reprogramming mitochondrial dynamics in fibroblasts.

Journal: Free radical biology & medicine

Published: March 15, 2026

H3K18 lactylation promotes allergic airway inflammation in asthma via a CSF1/CSF1R/MAPK autocrine axis.

H3K18 lactylation promotes allergic airway inflammation in asthma via a CSF1/CSF1R/MAPK autocrine axis.

Journal: Respiratory research

Published: February 03, 2026

5-methoxyindole-3-carboxaldehyde attenuates alveolar type II epithelial cell senescence induced by exposure to polystyrene microplastics in pulmonary fibrosis via PI3K/AKT/mTOR signaling pathway.

5-methoxyindole-3-carboxaldehyde attenuates alveolar type II epithelial cell senescence induced by exposure to polystyrene microplastics in pulmonary fibrosis via PI3K/AKT/mTOR signaling pathway.

Journal: Ecotoxicology and environmental safety

Published: September 21, 2025

Diagnostic model and identification of IGFBP2 as an aging-related biomarker and therapeutic target for systemic sclerosis-associated interstitial lung disease through integrated bioinformatics and machine learning approaches.

Diagnostic model and identification of IGFBP2 as an aging-related biomarker and therapeutic target for systemic sclerosis-associated interstitial lung disease through integrated bioinformatics and machine learning approaches.

Journal: International journal of surgery (London, England)

Published: September 01, 2025

Bacterial cellulose repairs asthmatic epithelial injury by reprogramming CDS1-mediated phosphatidylinositol metabolism to inhibit PI3K/AKT signaling.

Bacterial cellulose repairs asthmatic epithelial injury by reprogramming CDS1-mediated phosphatidylinositol metabolism to inhibit PI3K/AKT signaling.

Journal: International journal of biological macromolecules

Published: July 31, 2025

Disruption of the Clock Component BMAL1 in HDM-induced Asthma Causes GC Resistance in Airway Epithelial Cells by Regulating GR Phosphorylation through the DUSP4-p38MAPK Pathway.

Disruption of the Clock Component BMAL1 in HDM-induced Asthma Causes GC Resistance in Airway Epithelial Cells by Regulating GR Phosphorylation through the DUSP4-p38MAPK Pathway.

Journal: International journal of biological sciences

Published: June 13, 2025

Bioinformatics-based identification of mirdametinib as a potential therapeutic target for idiopathic pulmonary fibrosis associated with endoplasmic reticulum stress.

Bioinformatics-based identification of mirdametinib as a potential therapeutic target for idiopathic pulmonary fibrosis associated with endoplasmic reticulum stress.

Journal: Naunyn-Schmiedeberg's archives of pharmacology

Published: January 03, 2025

Preclinical models of immune checkpoint inhibitors-related interstitial pneumonia for anti-PD1 tumor immunotherapy.

Preclinical models of immune checkpoint inhibitors-related interstitial pneumonia for anti-PD1 tumor immunotherapy.

Journal: Immunobiology

Published: November 11, 2024

The association between body roundness index and asthma risk in middle-aged and elderly people: a retrospective cohort study based on CHARLS database.

The association between body roundness index and asthma risk in middle-aged and elderly people: a retrospective cohort study based on CHARLS database.

Journal: BMC pulmonary medicine

Published: September 21, 2024

Indole-3-acetic acid attenuates pulmonary fibrosis by modulating lung microbiota, inhibiting fibroblast activation, and alleviating alveolar epithelial cell senescence.

Indole-3-acetic acid attenuates pulmonary fibrosis by modulating lung microbiota, inhibiting fibroblast activation, and alleviating alveolar epithelial cell senescence.

Journal: Life sciences

Published: August 21, 2024

Tetrandrine Alleviates Pulmonary Fibrosis by Modulating Lung Microbiota-Derived Metabolism and Ameliorating Alveolar Epithelial Cell Senescence.

Tetrandrine Alleviates Pulmonary Fibrosis by Modulating Lung Microbiota-Derived Metabolism and Ameliorating Alveolar Epithelial Cell Senescence.

Journal: Phytotherapy research : PTR

Published: July 30, 2024

Showing 1-12 of 13

Last Updated: 09/26/2026