Qingke Duan
Union Hospital
Wuhan, 430022, China
Wuhan, 430022, China
Last Updated: 09/26/2026
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Found 10 publications
Interaction between m6A and YAP1 mechanotransduction pathways is essential for mechanical memory and matrix remodeling in pancreatic cancer.
Journal: International journal of biological sciences
Published: September 16, 2025
Neoadjuvant immunochemotherapy versus neoadjuvant immunoradiotherapy in locally advanced oral squamous cell carcinoma.
Journal: Frontiers in immunology
Published: January 20, 2025
Correction: Combination chemotherapy of valproic acid (VPA) and gemcitabine regulates STAT3/Bmi1 pathway to differentially potentiate the motility of pancreatic cancer cells.
Journal: Cell & bioscience
Published: October 26, 2023
ROS/KRAS/AMPK Signaling Contributes to Gemcitabine-Induced Stem-like Cell Properties in Pancreatic Cancer.
Journal: Molecular therapy oncolytics
Published: June 22, 2019
Combination chemotherapy of valproic acid (VPA) and gemcitabine regulates STAT3/Bmi1 pathway to differentially potentiate the motility of pancreatic cancer cells.
Journal: Cell & bioscience
Published: March 12, 2019
High glucose promotes pancreatic cancer cells to escape from immune surveillance via AMPK-Bmi1-GATA2-MICA/B pathway.
Journal: Journal of experimental & clinical cancer research : CR
Published: December 27, 2018
Mechanistic Evaluation and Translational Signature of Gemcitabine-induced Chemoresistance by Quantitative Phosphoproteomics Analysis with iTRAQ Labeling Mass Spectrometry.
Journal: Scientific reports
Published: April 19, 2017
Up-regulation of glycolysis promotes the stemness and EMT phenotypes in gemcitabine-resistant pancreatic cancer cells.
Journal: Journal of cellular and molecular medicine
Published: October 05, 2016
Bmi1 inhibition enhances the sensitivity of pancreatic cancer cells to gemcitabine.
Journal: Oncotarget
Published: September 25, 2015
Showing 1-10 of 10
Last Updated: 09/26/2026