Qiqi Guo
Nanjing, China
Last Updated: 09/26/2026
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Found 15 publications
BRD4 directs myofiber identity and metabolic adaptation through CHD4 cooperation.
Journal: Nature communications
Published: June 18, 2025
Glucose-Responsive PAGR1-Regulated Skeletal Muscle Gene Program Controls Systemic Glucose Homeostasis and Hepatic Metabolism.
Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: February 13, 2025
Enhancer regulator MLL4 controls skeletal muscle metabolic efficiency by limiting AMPK-mediated fuel catabolism.
Journal: Nature communications
Published: December 04, 2024
Imbalanced Skeletal Muscle Mitochondrial Proteostasis Causes Bone Loss.
Journal: Research (Washington, D.C.)
Published: April 21, 2024
Mitochondrial proteostasis stress in muscle drives a long-range protective response to alleviate dietary obesity independently of ATF4.
Journal: Science advances
Published: July 27, 2022
Proteolytic rewiring of mitochondria by LONP1 directs cell identity switching of adipocytes.
Journal: Nature cell biology
Published: June 26, 2022
FNIP1 regulates adipocyte browning and systemic glucose homeostasis in mice by shaping intracellular calcium dynamics.
Journal: The Journal of experimental medicine
Published: December 14, 2021
IRE1α regulates skeletal muscle regeneration through Myostatin mRNA decay.
Journal: The Journal of clinical investigation
Published: July 20, 2021
Disuse-associated loss of the protease LONP1 in muscle impairs mitochondrial function and causes reduced skeletal muscle mass and strength.
Journal: Nature communications
Published: April 14, 2021
Histone methyltransferase MLL4 controls myofiber identity and muscle performance through MEF2 interaction.
Journal: The Journal of clinical investigation
Published: January 08, 2020
Last Updated: 09/26/2026