Qiudong Zhao

225 Changhai Road
Shanghai, 200438, China

Last Updated: 09/26/2026

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Found 24 publications

Glycochenodeoxycholic acid promotes hepatocarcinogenesis by inducing hepatic progenitor cell differentiation into cancer-associated fibroblasts via sphingosine-1-phosphate receptor 2 signalling.

Glycochenodeoxycholic acid promotes hepatocarcinogenesis by inducing hepatic progenitor cell differentiation into cancer-associated fibroblasts via sphingosine-1-phosphate receptor 2 signalling.

Journal: Biomarker research

Published: June 08, 2025

ERG mediates the differentiation of hepatic progenitor cells towards immunosuppressive PDGFRα+ cancer-associated fibroblasts during hepatocarcinogenesis.

ERG mediates the differentiation of hepatic progenitor cells towards immunosuppressive PDGFRα+ cancer-associated fibroblasts during hepatocarcinogenesis.

Journal: Cell death & disease

Published: September 05, 2024

GPNMB+ Gal-3+ hepatic parenchymal cells promote immunosuppression and hepatocellular carcinogenesis.

GPNMB+ Gal-3+ hepatic parenchymal cells promote immunosuppression and hepatocellular carcinogenesis.

Journal: The EMBO journal

Published: March 20, 2023

TWEAK Signaling-Induced ID1 Expression Drives Malignant Transformation of Hepatic Progenitor Cells During Hepatocarcinogenesis.

TWEAK Signaling-Induced ID1 Expression Drives Malignant Transformation of Hepatic Progenitor Cells During Hepatocarcinogenesis.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: January 16, 2023

Immunosuppressive CD10+ALPL+ neutrophils promote resistance to anti-PD-1 therapy in HCC by mediating irreversible exhaustion of T cells.

Immunosuppressive CD10+ALPL+ neutrophils promote resistance to anti-PD-1 therapy in HCC by mediating irreversible exhaustion of T cells.

Journal: Journal of hepatology

Published: October 25, 2022

LAPTM4B-YAP loop feedback amplification enhances the stemness of hepatocellular carcinoma.

LAPTM4B-YAP loop feedback amplification enhances the stemness of hepatocellular carcinoma.

Journal: iScience

Published: September 23, 2022

AIF1 + CSF1R + MSCs, induced by TNF-α, act to generate an inflammatory microenvironment and promote hepatocarcinogenesis.

AIF1 + CSF1R + MSCs, induced by TNF-α, act to generate an inflammatory microenvironment and promote hepatocarcinogenesis.

Journal: Hepatology (Baltimore, Md.)

Published: April 28, 2022

Single cell transcriptional diversity and intercellular crosstalk of human liver cancer.

Single cell transcriptional diversity and intercellular crosstalk of human liver cancer.

Journal: Cell death & disease

Published: August 12, 2021

Oncostatin M promotes hepatic progenitor cell activation and hepatocarcinogenesis via macrophage-derived tumor necrosis factor-α.

Oncostatin M promotes hepatic progenitor cell activation and hepatocarcinogenesis via macrophage-derived tumor necrosis factor-α.

Journal: Cancer letters

Published: March 23, 2021

The stemness of hepatocytes is maintained by high levels of lipopolysaccharide via YAP1 activation.

The stemness of hepatocytes is maintained by high levels of lipopolysaccharide via YAP1 activation.

Journal: Stem cell research & therapy

Published: January 18, 2021

Correction: Kupffer cells-dependent inflammation in the injured liver increases recruitment of mesenchymal stem cells in aging mice.

Correction: Kupffer cells-dependent inflammation in the injured liver increases recruitment of mesenchymal stem cells in aging mice.

Journal: Oncotarget

Published: November 04, 2020

A TNFR2-hnRNPK Axis Promotes Primary Liver Cancer Development via Activation of YAP Signaling in Hepatic Progenitor Cells.

A TNFR2-hnRNPK Axis Promotes Primary Liver Cancer Development via Activation of YAP Signaling in Hepatic Progenitor Cells.

Journal: Cancer research

Published: September 18, 2020

Showing 1-12 of 24

Last Updated: 09/26/2026