Quentin Bazot

London, ENG SE19RT, United Kingdom

Last Updated: 09/26/2026

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Found 15 publications

The manufacture of AAV for gene therapy applications using a closed, semi-automated hollow-fiber bioreactor.

The manufacture of AAV for gene therapy applications using a closed, semi-automated hollow-fiber bioreactor.

Journal: Molecular therapy. Methods & clinical development

Published: November 20, 2024

Phosphodiesterase-induced cAMP degradation restricts hepatitis B virus infection.

Phosphodiesterase-induced cAMP degradation restricts hepatitis B virus infection.

Journal: Philosophical transactions of the Royal Society of London. Series B, Biological sciences

Published: April 09, 2019

Requirement for PRC1 subunit BMI1 in host gene activation by Epstein-Barr virus protein EBNA3C.

Requirement for PRC1 subunit BMI1 in host gene activation by Epstein-Barr virus protein EBNA3C.

Journal: Nucleic acids research

Published: September 14, 2018

Epstein-Barr Virus Nuclear Antigen 3C Inhibits Expression of COBLL1 and the ADAM28-ADAMDEC1 Locus via Interaction with the Histone Lysine Demethylase KDM2B.

Epstein-Barr Virus Nuclear Antigen 3C Inhibits Expression of COBLL1 and the ADAM28-ADAMDEC1 Locus via Interaction with the Histone Lysine Demethylase KDM2B.

Journal: Journal of virology

Published: August 08, 2018

Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.

Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.

Journal: Journal of virology

Published: November 03, 2017

Epstein-Barr virus nuclear antigen 1 interacts with regulator of chromosome condensation 1 dynamically throughout the cell cycle.

Epstein-Barr virus nuclear antigen 1 interacts with regulator of chromosome condensation 1 dynamically throughout the cell cycle.

Journal: The Journal of general virology

Published: March 13, 2017

EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.

EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.

Journal: PLoS biology

Published: January 12, 2017

Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection.

Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection.

Journal: eLife

Published: October 19, 2016

Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression.

Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression.

Journal: Nucleic acids research

Published: August 02, 2016

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.

Journal: PLoS pathogens

Published: October 12, 2015

The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells.

The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells.

Journal: Current topics in microbiology and immunology

Published: October 03, 2015

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.

Journal: PLoS pathogens

Published: January 30, 2015

Showing 1-12 of 15

Last Updated: 09/26/2026