Rafael M. Da Costa

Ribeirao Preto, SP, Brazil

Last Updated: 09/26/2026

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Found 12 publications

Elastase-2 deletion prevents vascular remodeling and abdominal aortic aneurysm development in a mice model induced by angiotensin II.

Elastase-2 deletion prevents vascular remodeling and abdominal aortic aneurysm development in a mice model induced by angiotensin II.

Journal: Vascular pharmacology
Published: June 16, 2025

NADPH oxidase 4-derived hydrogen peroxide counterbalances testosterone-induced endothelial dysfunction and migration.

NADPH oxidase 4-derived hydrogen peroxide counterbalances testosterone-induced endothelial dysfunction and migration.

Journal: American journal of physiology. Endocrinology and metabolism
Published: May 01, 2024

Melatonin prevents overproduction of reactive oxygen species and vascular dysfunction induced by cyclophosphamide.

Melatonin prevents overproduction of reactive oxygen species and vascular dysfunction induced by cyclophosphamide.

Journal: Life sciences
Published: October 30, 2023

Alpha 1-acid glycoprotein is upregulated in severe COVID-19 patients and decreases neutrophil NETs in SARS-CoV-2 infection.

Alpha 1-acid glycoprotein is upregulated in severe COVID-19 patients and decreases neutrophil NETs in SARS-CoV-2 infection.

Journal: Cytokine
Published: June 26, 2023

Aryl hydrocarbon receptor (AhR) activation contributes to high-fat diet-induced vascular dysfunction.

Aryl hydrocarbon receptor (AhR) activation contributes to high-fat diet-induced vascular dysfunction.

Journal: British journal of pharmacology
Published: April 29, 2021

Supraphysiological Levels of Testosterone Induce Vascular Dysfunction via Activation of the NLRP3 Inflammasome.

Supraphysiological Levels of Testosterone Induce Vascular Dysfunction via Activation of the NLRP3 Inflammasome.

Journal: Frontiers in immunology
Published: March 01, 2020

Nrf2 as a Potential Mediator of Cardiovascular Risk in Metabolic Diseases.

Nrf2 as a Potential Mediator of Cardiovascular Risk in Metabolic Diseases.

Journal: Frontiers in pharmacology
Published: November 07, 2018

Novel glucopyranoside C2-derived 1,2,3-triazoles displaying selective inhibition of O-GlcNAcase (OGA).

Novel glucopyranoside C2-derived 1,2,3-triazoles displaying selective inhibition of O-GlcNAcase (OGA).

Journal: Carbohydrate research
Published: September 21, 2018

Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism.

Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism.

Journal: Life sciences
Published: July 16, 2018

Increased O-GlcNAcylation of Endothelial Nitric Oxide Synthase Compromises the Anti-contractile Properties of Perivascular Adipose Tissue in Metabolic Syndrome.

Increased O-GlcNAcylation of Endothelial Nitric Oxide Synthase Compromises the Anti-contractile Properties of Perivascular Adipose Tissue in Metabolic Syndrome.

Journal: Frontiers in physiology
Published: December 07, 2017

Increased mitochondrial ROS generation mediates the loss of the anti-contractile effects of perivascular adipose tissue in high-fat diet obese mice.

Increased mitochondrial ROS generation mediates the loss of the anti-contractile effects of perivascular adipose tissue in high-fat diet obese mice.

Journal: British journal of pharmacology
Published: August 17, 2016

The vasorelaxant effect of gallic acid involves endothelium-dependent and -independent mechanisms.

The vasorelaxant effect of gallic acid involves endothelium-dependent and -independent mechanisms.

Journal: Vascular pharmacology
Published: September 16, 2015
Showing 1-12 of 12

Last Updated: 09/26/2026