Rahma Abdi-Hassan
Paris, 75019, France
Last Updated: 09/26/2026
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Found 15 publications
Author Correction: Organophosphorus diisopropylfluorophosphate (DFP) intoxication in zebrafish larvae causes behavioral defects, neuronal hyperexcitation and neuronal death.
Journal: Scientific reports
Published: March 22, 2021
Correction to: Surfen and oxalyl surfen decrease tau hyperphosphorylation and mitigate neuron deficits in vivo in a zebrafish model of tauopathy.
Journal: Translational neurodegeneration
Published: December 21, 2020
Damage-Induced Calcium Signaling and Reactive Oxygen Species Mediate Macrophage Activation in Zebrafish.
Journal: Frontiers in immunology
Published: December 01, 2020
Diisopropylfluorophosphate-induced status epilepticus drives complex glial cell phenotypes in adult male mice.
Journal: Neurobiology of disease
Published: September 14, 2020
A Fast, Simple, and Affordable Technique to Measure Oxygen Consumption in Living Zebrafish Embryos.
Journal: Zebrafish
Published: May 05, 2020
Organophosphorus diisopropylfluorophosphate (DFP) intoxication in zebrafish larvae causes behavioral defects, neuronal hyperexcitation and neuronal death.
Journal: Scientific reports
Published: March 05, 2020
Neurons Expressing Pathological Tau Protein Trigger Dramatic Changes in Microglial Morphology and Dynamics.
Journal: Frontiers in neuroscience
Published: August 07, 2019
Decreased microglial Wnt/β-catenin signalling drives microglial pro-inflammatory activation in the developing brain.
Journal: Brain : a journal of neurology
Published: October 23, 2018
γ-Aminobutyric acid receptor alpha 1 subunit loss of function causes genetic generalized epilepsy by impairing inhibitory network neurodevelopment.
Journal: Epilepsia
Published: June 29, 2018
Last Updated: 09/26/2026