Ray C. Bartolo

Parkville, VIC, Australia

Last Updated: 09/26/2026

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Found 11 publications

SFPQ-ABL1 and BCR-ABL1 use different signaling networks to drive B-cell acute lymphoblastic leukemia.

SFPQ-ABL1 and BCR-ABL1 use different signaling networks to drive B-cell acute lymphoblastic leukemia.

Journal: Blood advances
Published: September 02, 2021

Cotargeting BCL-2 and MCL-1 in high-risk B-ALL.

Cotargeting BCL-2 and MCL-1 in high-risk B-ALL.

Journal: Blood advances
Published: December 30, 2019

The application of RNA sequencing for the diagnosis and genomic classification of pediatric acute lymphoblastic leukemia.

The application of RNA sequencing for the diagnosis and genomic classification of pediatric acute lymphoblastic leukemia.

Journal: Blood advances
Published: September 20, 2019

Targeted therapy and disease monitoring in CNTRL-FGFR1-driven leukaemia.

Targeted therapy and disease monitoring in CNTRL-FGFR1-driven leukaemia.

Journal: Pediatric blood & cancer
Published: January 07, 2019

Clinker: visualizing fusion genes detected in RNA-seq data.

Clinker: visualizing fusion genes detected in RNA-seq data.

Journal: GigaScience
Published: January 13, 2018

Disordered clusters of Bak dimers rupture mitochondria during apoptosis.

Disordered clusters of Bak dimers rupture mitochondria during apoptosis.

Journal: eLife
Published: July 25, 2016

Functionally distinct roles for different miR-155 expression levels through contrasting effects on gene expression, in acute myeloid leukaemia.

Functionally distinct roles for different miR-155 expression levels through contrasting effects on gene expression, in acute myeloid leukaemia.

Journal: Leukemia
Published: January 03, 2016

Apoptotic pore formation is associated with in-plane insertion of Bak or Bax central helices into the mitochondrial outer membrane.

Apoptotic pore formation is associated with in-plane insertion of Bak or Bax central helices into the mitochondrial outer membrane.

Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: September 18, 2014

Dissociation of Bak α1 helix from the core and latch domains is required for apoptosis.

Dissociation of Bak α1 helix from the core and latch domains is required for apoptosis.

Journal: Nature communications
Published: July 27, 2014

Bak core and latch domains separate during activation, and freed core domains form symmetric homodimers.

Bak core and latch domains separate during activation, and freed core domains form symmetric homodimers.

Journal: Molecular cell
Published: May 16, 2014

Bak apoptotic function is not directly regulated by phosphorylation.

Bak apoptotic function is not directly regulated by phosphorylation.

Journal: Cell death & disease
Published: January 11, 2013
Showing 1-11 of 11

Last Updated: 09/26/2026