Rene Arnaud-St

Montreal, QC, Canada

Last Updated: 09/26/2026

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Found 61 publications

The 24,25(OH)2D3-TLCD3B signaling complex and reproductive function in mice and men.

The 24,25(OH)2D3-TLCD3B signaling complex and reproductive function in mice and men.

Journal: The Journal of steroid biochemistry and molecular biology
Published: April 01, 2026

The 24,25(OH)2D3-TLCD3B signaling complex and reproductive function in mice and men.

The 24,25(OH)2D3-TLCD3B signaling complex and reproductive function in mice and men.

Journal: The Journal of steroid biochemistry and molecular biology
Published: April 01, 2026

Inducible kidney-selective knockdown of Cyp27b1 does not compromise calcium and bone homeostasis.

Inducible kidney-selective knockdown of Cyp27b1 does not compromise calcium and bone homeostasis.

Journal: Journal of endocrinological investigation
Published: July 29, 2025

CYP24 inhibition as a therapeutic target in FGF23-mediated renal phosphate wasting disorders.

CYP24 inhibition as a therapeutic target in FGF23-mediated renal phosphate wasting disorders.

Journal: The Journal of clinical investigation
Published: April 15, 2025

Vitamin D stimulates Il-15 synthesis in rodent muscle.

Vitamin D stimulates Il-15 synthesis in rodent muscle.

Journal: Biochemistry and biophysics reports
Published: July 17, 2024

Loss of 24-hydroxylated catabolism increases calcitriol and fibroblast growth factor 23 and alters calcium and phosphate metabolism in fetal mice.

Loss of 24-hydroxylated catabolism increases calcitriol and fibroblast growth factor 23 and alters calcium and phosphate metabolism in fetal mice.

Journal: JBMR plus
Published: January 15, 2024

Maternal loss of 24-hydroxylase causes increased intestinal calcium absorption and hypercalcemia during pregnancy but reduced skeletal resorption during lactation in mice.

Maternal loss of 24-hydroxylase causes increased intestinal calcium absorption and hypercalcemia during pregnancy but reduced skeletal resorption during lactation in mice.

Journal: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
Published: December 13, 2023

Infantile hypercalcemia type 1 (HCINF1): a rare disease resulting in nephrolithiasis and nephrocalcinosis caused by mutations in the vitamin D catabolic enzyme, CYP24A1.

Infantile hypercalcemia type 1 (HCINF1): a rare disease resulting in nephrolithiasis and nephrocalcinosis caused by mutations in the vitamin D catabolic enzyme, CYP24A1.

Journal: Journal of endocrinological investigation
Published: December 12, 2023

Loss of maternal calcitriol reversibly alters early offspring growth and skeletal development in mice.

Loss of maternal calcitriol reversibly alters early offspring growth and skeletal development in mice.

Journal: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
Published: November 12, 2023

USP53 Regulates Bone Homeostasis by Controlling Rankl Expression in Osteoblasts and Bone Marrow Adipocytes.

USP53 Regulates Bone Homeostasis by Controlling Rankl Expression in Osteoblasts and Bone Marrow Adipocytes.

Journal: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
Published: August 16, 2022

Vitamin D and Diseases of Mineral Homeostasis: A Cyp24a1 R396W Humanized Preclinical Model of Infantile Hypercalcemia Type 1.

Vitamin D and Diseases of Mineral Homeostasis: A Cyp24a1 R396W Humanized Preclinical Model of Infantile Hypercalcemia Type 1.

Journal: Nutrients
Published: July 22, 2022

NACA and LRP6 Are Part of a Common Genetic Pathway Necessary for Full Anabolic Response to Intermittent PTH.

NACA and LRP6 Are Part of a Common Genetic Pathway Necessary for Full Anabolic Response to Intermittent PTH.

Journal: International journal of molecular sciences
Published: December 13, 2021
Showing 1-12 of 61

Last Updated: 09/26/2026