Renee I. Seinstra

Groningen, GR 9713AV, Netherlands

Last Updated: 09/26/2026

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Found 20 publications

Condensate-Driven Triglyceride Depletion Links α-Synuclein to Mitochondrial Dysfunction.

Condensate-Driven Triglyceride Depletion Links α-Synuclein to Mitochondrial Dysfunction.

Journal: bioRxiv : the preprint server for biology

Published: November 24, 2025

Ferroptosis inhibition protects against α-synuclein-related neuronal cell death.

Ferroptosis inhibition protects against α-synuclein-related neuronal cell death.

Journal: Cell death & disease

Published: March 26, 2025

In Vivo Nanodiamond Quantum Sensing of Free Radicals in Caenorhabditis elegans Models.

In Vivo Nanodiamond Quantum Sensing of Free Radicals in Caenorhabditis elegans Models.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: October 03, 2024

Rebalancing the motor circuit restores movement in a Caenorhabditis elegans model for TDP-43 toxicity.

Rebalancing the motor circuit restores movement in a Caenorhabditis elegans model for TDP-43 toxicity.

Journal: Cell reports

Published: July 04, 2023

Neuronal overexpression of human TDP-43 in Caenorhabditis elegans causes a range of sensorimotor phenotypes.

Neuronal overexpression of human TDP-43 in Caenorhabditis elegans causes a range of sensorimotor phenotypes.

Journal: microPublication biology

Published: February 10, 2023

Neuronal overexpression of hTDP-43 in Caenorhabditis elegans mimics the cellular pathology commonly observed in TDP-43 proteinopathies.

Neuronal overexpression of hTDP-43 in Caenorhabditis elegans mimics the cellular pathology commonly observed in TDP-43 proteinopathies.

Journal: microPublication biology

Published: February 10, 2023

Neuronal overexpression of hTDP-43 in Caenorhabditis elegans impairs motor function.

Neuronal overexpression of hTDP-43 in Caenorhabditis elegans impairs motor function.

Journal: microPublication biology

Published: February 10, 2023

Neuronal overexpression of hTDP-43 in Caenorhabditis elegans impairs different neuronally controlled behaviors and decreases fecundity.

Neuronal overexpression of hTDP-43 in Caenorhabditis elegans impairs different neuronally controlled behaviors and decreases fecundity.

Journal: microPublication biology

Published: February 10, 2023

The cellular modifier MOAG-4/SERF drives amyloid formation through charge complementation.

The cellular modifier MOAG-4/SERF drives amyloid formation through charge complementation.

Journal: The EMBO journal

Published: December 23, 2020

Comparative Studies in the A30P and A53T α-Synuclein C. elegans Strains to Investigate the Molecular Origins of Parkinson's Disease.

Comparative Studies in the A30P and A53T α-Synuclein C. elegans Strains to Investigate the Molecular Origins of Parkinson's Disease.

Journal: Frontiers in cell and developmental biology

Published: April 21, 2020

Assessing motor-related phenotypes of Caenorhabditis elegans with the wide field-of-view nematode tracking platform.

Assessing motor-related phenotypes of Caenorhabditis elegans with the wide field-of-view nematode tracking platform.

Journal: Nature protocols

Published: October 17, 2019

C. elegans as a Model for Synucleinopathies and Other Neurodegenerative Diseases: Tools and Techniques.

C. elegans as a Model for Synucleinopathies and Other Neurodegenerative Diseases: Tools and Techniques.

Journal: Methods in molecular biology (Clifton, N.J.)

Published: February 17, 2019

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Last Updated: 09/26/2026