Ricardo Amorim

Coimbra, 3000548, Portugal

Last Updated: 09/26/2026

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Found 28 publications

Mitochondria-targeted antioxidant AntiOxBEN2 prevents metabolic dysfunction-associated steatotic liver disease (MASLD) by enhancing fatty acid oxidation and mitochondrial bioenergetics.

Mitochondria-targeted antioxidant AntiOxBEN2 prevents metabolic dysfunction-associated steatotic liver disease (MASLD) by enhancing fatty acid oxidation and mitochondrial bioenergetics.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: September 08, 2025

Optimising rare tumour risk syndromes care: clinical insights and the PREVENTABLE socioeconomic framework.

Optimising rare tumour risk syndromes care: clinical insights and the PREVENTABLE socioeconomic framework.

Journal: European journal of human genetics : EJHG

Published: June 23, 2025

Chemical Characterization and Differential Lipid-Modulating Effects of Selected Plant Extracts from Côa Valley (Portugal) in a Cell Model for Liver Steatosis.

Chemical Characterization and Differential Lipid-Modulating Effects of Selected Plant Extracts from Côa Valley (Portugal) in a Cell Model for Liver Steatosis.

Journal: Pharmaceuticals (Basel, Switzerland)

Published: December 11, 2024

Metabolic profile of human non-small cell lung cancer cells through combined 13C and 2H NMR.

Metabolic profile of human non-small cell lung cancer cells through combined 13C and 2H NMR.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: November 30, 2024

The mitochondriotropic antioxidants AntiOxBEN2 and AntiOxCIN4 are structurally-similar but differentially alter energy homeostasis in human skin fibroblasts.

The mitochondriotropic antioxidants AntiOxBEN2 and AntiOxCIN4 are structurally-similar but differentially alter energy homeostasis in human skin fibroblasts.

Journal: Biochimica et biophysica acta. Bioenergetics

Published: November 12, 2024

Decreasing the burden of non-alcoholic fatty liver disease: From therapeutic targets to drug discovery opportunities.

Decreasing the burden of non-alcoholic fatty liver disease: From therapeutic targets to drug discovery opportunities.

Journal: European journal of medicinal chemistry

Published: May 30, 2024

Nucleotide metabolism in cancer cells fuels a UDP-driven macrophage cross-talk, promoting immunosuppression and immunotherapy resistance.

Nucleotide metabolism in cancer cells fuels a UDP-driven macrophage cross-talk, promoting immunosuppression and immunotherapy resistance.

Journal: Nature cancer

Published: January 05, 2024

Mitochondria dysfunction induced by decyl-TPP mitochondriotropic antioxidant based on caffeic acid AntiOxCIN6 sensitizes cisplatin lung anticancer therapy due to a remodeling of energy metabolism.

Mitochondria dysfunction induced by decyl-TPP mitochondriotropic antioxidant based on caffeic acid AntiOxCIN6 sensitizes cisplatin lung anticancer therapy due to a remodeling of energy metabolism.

Journal: Biochemical pharmacology

Published: September 27, 2023

From Non-Alcoholic Fatty Liver to Hepatocellular Carcinoma: A Story of (Mal)Adapted Mitochondria.

From Non-Alcoholic Fatty Liver to Hepatocellular Carcinoma: A Story of (Mal)Adapted Mitochondria.

Journal: Biology

Published: March 10, 2023

Mitochondria-targeted anti-oxidant AntiOxCIN4 improved liver steatosis in Western diet-fed mice by preventing lipid accumulation due to upregulation of fatty acid oxidation, quality control mechanism and antioxidant defense systems.

Mitochondria-targeted anti-oxidant AntiOxCIN4 improved liver steatosis in Western diet-fed mice by preventing lipid accumulation due to upregulation of fatty acid oxidation, quality control mechanism and antioxidant defense systems.

Journal: Redox biology

Published: April 27, 2022

RIPK3 dampens mitochondrial bioenergetics and lipid droplet dynamics in metabolic liver disease.

RIPK3 dampens mitochondrial bioenergetics and lipid droplet dynamics in metabolic liver disease.

Journal: Hepatology (Baltimore, Md.)

Published: March 26, 2022

Mitochondriotropic antioxidant based on caffeic acid AntiOxCIN4 activates Nrf2-dependent antioxidant defenses and quality control mechanisms to antagonize oxidative stress-induced cell damage.

Mitochondriotropic antioxidant based on caffeic acid AntiOxCIN4 activates Nrf2-dependent antioxidant defenses and quality control mechanisms to antagonize oxidative stress-induced cell damage.

Journal: Free radical biology & medicine

Published: September 06, 2021

Showing 1-12 of 28

Last Updated: 09/26/2026