Rifeng Gao

No. 88 Jiefang Road
Hangzhou, 310009, China

Last Updated: 09/26/2026

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Found 42 publications

Loss of eIF3m aggravates myocardial ischemia/reperfusion injury via enhancing nitrative damage and inhibiting glycolytic reprogramming.

Loss of eIF3m aggravates myocardial ischemia/reperfusion injury via enhancing nitrative damage and inhibiting glycolytic reprogramming.

Journal: Science bulletin

Published: January 10, 2026

CCL2 drives macrophage M2 polarization via CH25H-mediated metabolic reprogramming to ameliorate post-myocardial infarction remodeling.

CCL2 drives macrophage M2 polarization via CH25H-mediated metabolic reprogramming to ameliorate post-myocardial infarction remodeling.

Journal: Cellular signalling

Published: August 26, 2025

Young Regulatory T Cell-Derived Extracellular Vesicles Improve Mitochondrial Function and Angiogenesis and Suppress Inflammation in Senescent Brain and Heart.

Young Regulatory T Cell-Derived Extracellular Vesicles Improve Mitochondrial Function and Angiogenesis and Suppress Inflammation in Senescent Brain and Heart.

Journal: Journal of neurochemistry

Published: July 28, 2025

Targeting Endothelial KDM5A to Attenuate Aging and Ameliorate Age-Associated Metabolic Abnormalities.

Targeting Endothelial KDM5A to Attenuate Aging and Ameliorate Age-Associated Metabolic Abnormalities.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: July 08, 2025

Homotransplantation of Engineered Macrophages with Surface-Modified Nanomedicines for Mitigating Myocardial Ischemia-Reperfusion Injury.

Homotransplantation of Engineered Macrophages with Surface-Modified Nanomedicines for Mitigating Myocardial Ischemia-Reperfusion Injury.

Journal: ACS nano

Published: June 27, 2025

ALKBH5 Regulates Macrophage Senescence and Accelerates Atherosclerosis by Promoting CCL5 m6A Modification.

ALKBH5 Regulates Macrophage Senescence and Accelerates Atherosclerosis by Promoting CCL5 m6A Modification.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: April 03, 2025

Caspase-12 exhibits non-redundant functions in response to endoplasmic reticulum stress to promote GSDMD-mediated NETosis, leading to thoracic aortic dissection.

Caspase-12 exhibits non-redundant functions in response to endoplasmic reticulum stress to promote GSDMD-mediated NETosis, leading to thoracic aortic dissection.

Journal: Translational research : the journal of laboratory and clinical medicine

Published: December 09, 2024

CCL28 contributes to angiogenesis and cardiac repair through CCR10+ endothelial cells after myocardial infarction in male mice.

CCL28 contributes to angiogenesis and cardiac repair through CCR10+ endothelial cells after myocardial infarction in male mice.

Journal: Nature communications

Published: August 09, 2024

A Nanocapsule System Combats Aging by Inhibiting Age-Related Angiogenesis Deficiency and Glucolipid Metabolism Disorders.

A Nanocapsule System Combats Aging by Inhibiting Age-Related Angiogenesis Deficiency and Glucolipid Metabolism Disorders.

Journal: ACS nano

Published: August 01, 2024

SHEP1 alleviates cardiac ischemia reperfusion injury via targeting G3BP1 to regulate macrophage infiltration and inflammation.

SHEP1 alleviates cardiac ischemia reperfusion injury via targeting G3BP1 to regulate macrophage infiltration and inflammation.

Journal: Cell death & disease

Published: February 17, 2024

Selective inhibition of the NLRP3 inflammasome protects against acute ethanol-induced cardiotoxicity in an FBXL2-dependent manner.

Selective inhibition of the NLRP3 inflammasome protects against acute ethanol-induced cardiotoxicity in an FBXL2-dependent manner.

Journal: Acta biochimica et biophysica Sinica

Published: November 23, 2023

CCL11 released by GSDMD-mediated macrophage pyroptosis regulates angiogenesis after hindlimb ischemia.

CCL11 released by GSDMD-mediated macrophage pyroptosis regulates angiogenesis after hindlimb ischemia.

Journal: Cell death discovery

Published: September 02, 2023

Showing 1-12 of 42

Last Updated: 09/26/2026