Robert Stehle
Robert Koch Str. 39
Koeln, NW 50931, Germany
Koeln, NW 50931, Germany
Last Updated: 09/26/2026
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Found 25 publications
Phosphate rebinding induces force reversal via slow backward cycling of cross-bridges.
Journal: Frontiers in physiology
Published: August 06, 2024
De Novo Missense Mutations in TNNC1 and TNNI3 Causing Severe Infantile Cardiomyopathy Affect Myofilament Structure and Function and Are Modulated by Troponin Targeting Agents.
Journal: International journal of molecular sciences
Published: August 09, 2021
Phosphate has dual roles in cross-bridge kinetics in rabbit psoas single myofibrils.
Journal: The Journal of general physiology
Published: September 05, 2020
Hypertrophic and Dilated Cardiomyopathy-Associated Troponin T Mutations R130C and ΔK210 Oppositely Affect Length-Dependent Calcium Sensitivity of Force Generation.
Journal: Frontiers in physiology
Published: January 17, 2020
Infantile restrictive cardiomyopathy: cTnI-R170G/W impair the interplay of sarcomeric proteins and the integrity of thin filaments.
Journal: PloS one
Published: November 16, 2019
Cycling Cross-Bridges Contribute to Thin Filament Activation in Human Slow-Twitch Fibers.
Journal: Frontiers in physiology
Published: October 24, 2019
Cross-bridges in slow skeletal muscle: not only slow but also soft.
Journal: The Journal of physiology
Published: February 11, 2018
Erratum to: Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy.
Journal: Cellular and molecular life sciences : CMLS
Published: August 31, 2017
Kinetic coupling of phosphate release, force generation and rate-limiting steps in the cross-bridge cycle.
Journal: Journal of muscle research and cell motility
Published: July 05, 2017
Early signs of architectural and biomechanical failure in isolated myofibers and immortalized myoblasts from desmin-mutant knock-in mice.
Journal: Scientific reports
Published: January 10, 2017
Last Updated: 09/26/2026