Ross W. Milne

40 Ruskin Street, K1Y 4W7
Ottawa, ON, Canada

Last Updated: 09/26/2026

Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 14 publications

The Keap1-Nrf2 Stress Response Pathway Promotes Mitochondrial Hyperfusion Through Degradation of the Mitochondrial Fission Protein Drp1.

The Keap1-Nrf2 Stress Response Pathway Promotes Mitochondrial Hyperfusion Through Degradation of the Mitochondrial Fission Protein Drp1.

Journal: Antioxidants & redox signaling
Published: May 13, 2017

Methylglyoxal-derived advanced glycation end products contribute to negative cardiac remodeling and dysfunction post-myocardial infarction.

Methylglyoxal-derived advanced glycation end products contribute to negative cardiac remodeling and dysfunction post-myocardial infarction.

Journal: Basic research in cardiology
Published: February 21, 2017

Methylglyoxal-Induced Endothelial Cell Loss and Inflammation Contribute to the Development of Diabetic Cardiomyopathy.

Methylglyoxal-Induced Endothelial Cell Loss and Inflammation Contribute to the Development of Diabetic Cardiomyopathy.

Journal: Diabetes
Published: April 28, 2015

Apolipoprotein E Mediates Evasion From Hepatitis C Virus Neutralizing Antibodies.

Apolipoprotein E Mediates Evasion From Hepatitis C Virus Neutralizing Antibodies.

Journal: Gastroenterology
Published: September 24, 2014

Hyperglycemia inhibits cardiac stem cell-mediated cardiac repair and angiogenic capacity.

Hyperglycemia inhibits cardiac stem cell-mediated cardiac repair and angiogenic capacity.

Journal: Circulation
Published: September 10, 2014

Differential effects of glyoxalase 1 overexpression on diabetic atherosclerosis and renal dysfunction in streptozotocin-treated, apolipoprotein E-deficient mice.

Differential effects of glyoxalase 1 overexpression on diabetic atherosclerosis and renal dysfunction in streptozotocin-treated, apolipoprotein E-deficient mice.

Journal: Physiological reports
Published: June 13, 2014

Reducing methylglyoxal as a therapeutic target for diabetic heart disease.

Reducing methylglyoxal as a therapeutic target for diabetic heart disease.

Journal: Biochemical Society transactions
Published: March 21, 2014

Glyoxalase-1 overexpression in bone marrow cells reverses defective neovascularization in STZ-induced diabetic mice.

Glyoxalase-1 overexpression in bone marrow cells reverses defective neovascularization in STZ-induced diabetic mice.

Journal: Cardiovascular research
Published: November 22, 2013

Knockdown of glyoxalase 1 mimics diabetic nephropathy in nondiabetic mice.

Knockdown of glyoxalase 1 mimics diabetic nephropathy in nondiabetic mice.

Journal: Diabetes
Published: September 25, 2013

Role of renin-angiotensin system in activation of macrophages by modified lipoproteins.

Role of renin-angiotensin system in activation of macrophages by modified lipoproteins.

Journal: American journal of physiology. Heart and circulatory physiology
Published: September 03, 2013

Cathepsin G deficiency decreases complexity of atherosclerotic lesions in apolipoprotein E-deficient mice.

Cathepsin G deficiency decreases complexity of atherosclerotic lesions in apolipoprotein E-deficient mice.

Journal: American journal of physiology. Heart and circulatory physiology
Published: August 13, 2013

Insulin-degrading enzyme deficiency in bone marrow cells increases atherosclerosis in LDL receptor-deficient mice.

Insulin-degrading enzyme deficiency in bone marrow cells increases atherosclerosis in LDL receptor-deficient mice.

Journal: Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology
Published: January 14, 2013
Showing 1-12 of 14

Last Updated: 09/26/2026