Rui Appelberg

Porto, Portugal

Last Updated: 09/26/2026

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Found 32 publications

Advances on the Role and Applications of Interleukin-1 in Tuberculosis.

Advances on the Role and Applications of Interleukin-1 in Tuberculosis.

Journal: mBio

Published: November 23, 2021

IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on Mycobacterium avium-Induced Thymic Atrophy.

IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on Mycobacterium avium-Induced Thymic Atrophy.

Journal: Frontiers in immunology

Published: April 16, 2021

Deficiency in the glycosyltransferase Gcnt1 increases susceptibility to tuberculosis through a mechanism involving neutrophils.

Deficiency in the glycosyltransferase Gcnt1 increases susceptibility to tuberculosis through a mechanism involving neutrophils.

Journal: Mucosal immunology

Published: July 26, 2019

IFN-γ-Dependent Reduction of Erythrocyte Life Span Leads to Anemia during Mycobacterial Infection.

IFN-γ-Dependent Reduction of Erythrocyte Life Span Leads to Anemia during Mycobacterial Infection.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: April 03, 2019

Myeloid HIF-1α regulates pulmonary inflammation during experimental Mycobacterium tuberculosis infection.

Myeloid HIF-1α regulates pulmonary inflammation during experimental Mycobacterium tuberculosis infection.

Journal: Immunology

Published: January 04, 2019

TNF-Mediated Compensatory Immunity to Mycobacterium avium in the Absence of Macrophage Activation by IFN-γ.

TNF-Mediated Compensatory Immunity to Mycobacterium avium in the Absence of Macrophage Activation by IFN-γ.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: December 05, 2018

Innate IFN-γ-Producing Cells Developing in the Absence of IL-2 Receptor Common γ-Chain.

Innate IFN-γ-Producing Cells Developing in the Absence of IL-2 Receptor Common γ-Chain.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: October 07, 2016

Type I IFN Inhibits Alternative Macrophage Activation during Mycobacterium tuberculosis Infection and Leads to Enhanced Protection in the Absence of IFN-γ Signaling.

Type I IFN Inhibits Alternative Macrophage Activation during Mycobacterium tuberculosis Infection and Leads to Enhanced Protection in the Absence of IFN-γ Signaling.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: April 01, 2016

Delivery of LLKKK18 loaded into self-assembling hyaluronic acid nanogel for tuberculosis treatment.

Delivery of LLKKK18 loaded into self-assembling hyaluronic acid nanogel for tuberculosis treatment.

Journal: Journal of controlled release : official journal of the Controlled Release Society

Published: March 02, 2016

Antimicrobial peptides as novel anti-tuberculosis therapeutics.

Antimicrobial peptides as novel anti-tuberculosis therapeutics.

Journal: Biotechnology advances

Published: January 05, 2016

Studies in the mouse model identify strain variability as a major determinant of disease outcome in Leishmania infantum infection.

Studies in the mouse model identify strain variability as a major determinant of disease outcome in Leishmania infantum infection.

Journal: Parasites & vectors

Published: September 16, 2015

IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages.

IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages.

Journal: Frontiers in immunology

Published: June 16, 2015

Showing 1-12 of 32

Last Updated: 09/26/2026