Ruilong Peng

154 Anshan Road
Tianjin, China

Last Updated: 09/26/2026

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Found 17 publications

Neutrophil extracellular traps induce endothelial damage and exacerbate vasospasm in traumatic brain injury.

Neutrophil extracellular traps induce endothelial damage and exacerbate vasospasm in traumatic brain injury.

Journal: Theranostics
Published: April 14, 2025

Corrigendum to "Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury" [International Immunopharmacology 130 (2024) 111674].

Corrigendum to "Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury" [International Immunopharmacology 130 (2024) 111674].

Journal: International immunopharmacology
Published: March 27, 2025

Benzbromarone improves blood hypercoagulability after TBI by reducing phosphatidylserine externalization through inhibition of TMEM16F expression.

Benzbromarone improves blood hypercoagulability after TBI by reducing phosphatidylserine externalization through inhibition of TMEM16F expression.

Journal: Life sciences
Published: December 18, 2024

β2 integrin regulates neutrophil trans endothelial migration following traumatic brain injury.

β2 integrin regulates neutrophil trans endothelial migration following traumatic brain injury.

Journal: Cell communication and signaling : CCS
Published: November 08, 2024

Progesterone induces neuroprotection associated with immune/inflammatory modulation in experimental traumatic brain injury.

Progesterone induces neuroprotection associated with immune/inflammatory modulation in experimental traumatic brain injury.

Journal: Neuroreport
Published: March 25, 2024

CLEC7A regulates M2 macrophages to suppress the immune microenvironment and implies poorer prognosis of glioma.

CLEC7A regulates M2 macrophages to suppress the immune microenvironment and implies poorer prognosis of glioma.

Journal: Frontiers in immunology
Published: December 25, 2023

Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury.

Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury.

Journal: International immunopharmacology
Published: November 27, 2023

Iron overload enhances TBI-induced cardiac dysfunction by promoting ferroptosis and cardiac inflammation.

Iron overload enhances TBI-induced cardiac dysfunction by promoting ferroptosis and cardiac inflammation.

Journal: Biochemical and biophysical research communications
Published: July 23, 2023

NCOA4-mediated ferritinophagy aggravate intestinal oxidative stress and ferroptosis after traumatic brain injury.

NCOA4-mediated ferritinophagy aggravate intestinal oxidative stress and ferroptosis after traumatic brain injury.

Journal: Biochemical and biophysical research communications
Published: July 22, 2023

Brain-derived extracellular vesicles mediate traumatic brain injury associated multi-organ damage.

Brain-derived extracellular vesicles mediate traumatic brain injury associated multi-organ damage.

Journal: Biochemical and biophysical research communications
Published: April 02, 2023

Circulating extracellular vesicles from patients with traumatic brain injury induce cerebrovascular endothelial dysfunction.

Circulating extracellular vesicles from patients with traumatic brain injury induce cerebrovascular endothelial dysfunction.

Journal: Pharmacological research
Published: January 27, 2023

Abrocitinib Attenuates Microglia-Mediated Neuroinflammation after Traumatic Brain Injury via Inhibiting the JAK1/STAT1/NF-κB Pathway.

Abrocitinib Attenuates Microglia-Mediated Neuroinflammation after Traumatic Brain Injury via Inhibiting the JAK1/STAT1/NF-κB Pathway.

Journal: Cells
Published: September 16, 2022
Showing 1-12 of 17

Last Updated: 09/26/2026