Ruomeng Li

Fujian Key Laboratory Of Cognitive Function And Diseases
Fuzhou, China

Last Updated: 09/26/2026

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Found 15 publications

A Novel PROTAC Confers a Dual Benefit Against Amyloid and Tau Pathology in Alzheimer's Disease via DAPK1 Degradation.

A Novel PROTAC Confers a Dual Benefit Against Amyloid and Tau Pathology in Alzheimer's Disease via DAPK1 Degradation.

Journal: International journal of biological sciences

Published: January 14, 2026

Selective degradation of DAPK1 via a novel hydrophobic tagging attenuates tau pathology in Alzheimer's disease.

Selective degradation of DAPK1 via a novel hydrophobic tagging attenuates tau pathology in Alzheimer's disease.

Journal: Journal of advanced research

Published: August 09, 2025

Death-associated protein kinase 1-dependent SENP1 degradation increases tau SUMOylation and leads to cognitive dysfunction in a mouse model for tauopathy.

Death-associated protein kinase 1-dependent SENP1 degradation increases tau SUMOylation and leads to cognitive dysfunction in a mouse model for tauopathy.

Journal: Molecular neurodegeneration

Published: June 23, 2025

Design, synthesis, and biological evaluation of death-associated protein kinase 1 PROTACs.

Design, synthesis, and biological evaluation of death-associated protein kinase 1 PROTACs.

Journal: Bioorganic chemistry

Published: June 21, 2025

Saikosaponin C ameliorates tau-related pathology by modulating oxidative stress and MAPK axis in Alzheimer's disease.

Saikosaponin C ameliorates tau-related pathology by modulating oxidative stress and MAPK axis in Alzheimer's disease.

Journal: Journal of ethnopharmacology

Published: May 08, 2025

Zipper-interacting protein kinase mediates neuronal cell death and cognitive dysfunction in traumatic brain injury via regulating DEDD.

Zipper-interacting protein kinase mediates neuronal cell death and cognitive dysfunction in traumatic brain injury via regulating DEDD.

Journal: Cell death & disease

Published: September 12, 2024

Quantitative Proteomic and Phosphoproteomic Analyses Reveal a Role of Death-Associated Protein Kinase 1 in Regulating Hippocampal Synapse.

Quantitative Proteomic and Phosphoproteomic Analyses Reveal a Role of Death-Associated Protein Kinase 1 in Regulating Hippocampal Synapse.

Journal: Molecular neurobiology

Published: July 11, 2023

Activation of transient receptor potential vanilloid 1 ameliorates tau accumulation-induced synaptic damage and cognitive dysfunction via autophagy enhancement.

Activation of transient receptor potential vanilloid 1 ameliorates tau accumulation-induced synaptic damage and cognitive dysfunction via autophagy enhancement.

Journal: CNS neuroscience & therapeutics

Published: May 02, 2023

Death-associated protein kinase 1 phosphorylates MDM2 and inhibits its protein stability and function.

Death-associated protein kinase 1 phosphorylates MDM2 and inhibits its protein stability and function.

Journal: Archives of pharmacal research

Published: March 15, 2023

Ablation of Death-Associated Protein Kinase 1 Changes the Transcriptomic Profile and Alters Neural-Related Pathways in the Brain.

Ablation of Death-Associated Protein Kinase 1 Changes the Transcriptomic Profile and Alters Neural-Related Pathways in the Brain.

Journal: International journal of molecular sciences

Published: February 27, 2023

MiR-191-5p Attenuates Tau Phosphorylation, Aβ Generation, and Neuronal Cell Death by Regulating Death-Associated Protein Kinase 1.

MiR-191-5p Attenuates Tau Phosphorylation, Aβ Generation, and Neuronal Cell Death by Regulating Death-Associated Protein Kinase 1.

Journal: ACS chemical neuroscience

Published: December 01, 2022

miR-143-3p Inhibits Aberrant Tau Phosphorylation and Amyloidogenic Processing of APP by Directly Targeting DAPK1 in Alzheimer's Disease.

miR-143-3p Inhibits Aberrant Tau Phosphorylation and Amyloidogenic Processing of APP by Directly Targeting DAPK1 in Alzheimer's Disease.

Journal: International journal of molecular sciences

Published: May 31, 2022

Showing 1-12 of 15

Last Updated: 09/26/2026