Ruth Rollason
Bristol, ENG BS13NY, United Kingdom
Last Updated: 09/26/2026
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Found 11 publications
Pathogenic podocin variants exhibit distinct defects in trafficking, membrane organization, and degradation pathways.
Journal: European journal of cell biology
Published: May 30, 2025
Adeno-associated virus gene therapy prevents progression of kidney disease in genetic models of nephrotic syndrome.
Journal: Science translational medicine
Published: August 09, 2023
A small molecule chaperone rescues keratin-8 mediated trafficking of misfolded podocin to correct genetic Nephrotic Syndrome.
Journal: Kidney international
Published: March 03, 2023
TRPC6 Binds to and Activates Calpain, Independent of Its Channel Activity, and Regulates Podocyte Cytoskeleton, Cell Adhesion, and Motility.
Journal: Journal of the American Society of Nephrology : JASN
Published: July 16, 2018
Prolonged exposure of mouse and human podocytes to insulin induces insulin resistance through lysosomal and proteasomal degradation of the insulin receptor.
Journal: Diabetologia
Published: February 21, 2017
MAGI2 Mutations Cause Congenital Nephrotic Syndrome.
Journal: Journal of the American Society of Nephrology : JASN
Published: April 01, 2016
Disease causing mutations in inverted formin 2 regulate its binding to G-actin, F-actin capping protein (CapZ α-1) and profilin 2.
Journal: Bioscience reports
Published: October 01, 2015
CD317/tetherin is an organiser of membrane microdomains.
Journal: Journal of cell science
Published: February 05, 2013
Expression of HIV-1 Vpu leads to loss of the viral restriction factor CD317/Tetherin from lipid rafts and its enhanced lysosomal degradation.
Journal: PloS one
Published: October 16, 2012
Release of filamentous and spherical influenza A virus is not restricted by tetherin.
Journal: The Journal of general virology
Published: January 20, 2012
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Last Updated: 09/26/2026