Ryo Fujioka

Saitama, 3510198, Japan

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 11 publications

The Role of Neprilysin and Insulin-Degrading Enzyme in the Etiology of Sporadic Alzheimer's Disease.

The Role of Neprilysin and Insulin-Degrading Enzyme in the Etiology of Sporadic Alzheimer's Disease.

Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience

Published: November 11, 2024

The dopaminergic system promotes neprilysin-mediated degradation of amyloid-β in the brain.

The dopaminergic system promotes neprilysin-mediated degradation of amyloid-β in the brain.

Journal: Science signaling

Published: August 06, 2024

Tau Accumulation Induces Microglial State Alterations in Alzheimer's Disease Model Mice.

Tau Accumulation Induces Microglial State Alterations in Alzheimer's Disease Model Mice.

Journal: eNeuro

Published: June 12, 2024

Metabolic resistance of Aβ3pE-42, a target epitope of the anti-Alzheimer therapeutic antibody, donanemab.

Metabolic resistance of Aβ3pE-42, a target epitope of the anti-Alzheimer therapeutic antibody, donanemab.

Journal: Life science alliance

Published: February 08, 2024

The Icelandic Mutation (APP-A673T) Is Protective against Amyloid Pathology In Vivo.

The Icelandic Mutation (APP-A673T) Is Protective against Amyloid Pathology In Vivo.

Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience

Published: February 01, 2024

An isogenic panel of App knock-in mouse models: Profiling β-secretase inhibition and endosomal abnormalities.

An isogenic panel of App knock-in mouse models: Profiling β-secretase inhibition and endosomal abnormalities.

Journal: Science advances

Published: June 08, 2022

Increased CSF-decorin predicts brain pathological changes driven by Alzheimer's Aβ amyloidosis.

Increased CSF-decorin predicts brain pathological changes driven by Alzheimer's Aβ amyloidosis.

Journal: Acta neuropathologica communications

Published: March 10, 2022

A third-generation mouse model of Alzheimer's disease shows early and increased cored plaque pathology composed of wild-type human amyloid β peptide.

A third-generation mouse model of Alzheimer's disease shows early and increased cored plaque pathology composed of wild-type human amyloid β peptide.

Journal: The Journal of biological chemistry

Published: June 11, 2021

Correction for Takano et al., "Vital Role of the Calpain-Calpastatin System for Placental-Integrity-Dependent Embryonic Survival".

Correction for Takano et al., "Vital Role of the Calpain-Calpastatin System for Placental-Integrity-Dependent Embryonic Survival".

Journal: Molecular and cellular biology

Published: January 31, 2018

Introduction of pathogenic mutations into the mouse Psen1 gene by Base Editor and Target-AID.

Introduction of pathogenic mutations into the mouse Psen1 gene by Base Editor and Target-AID.

Journal: Nature communications

Published: January 27, 2018

Expression of Concern for Takano et al., "Vital Role of the Calpain-Calpastatin System for Placental-Integrity-Dependent Embryonic Survival".

Expression of Concern for Takano et al., "Vital Role of the Calpain-Calpastatin System for Placental-Integrity-Dependent Embryonic Survival".

Journal: Molecular and cellular biology

Published: November 15, 2017

Showing 1-11 of 11

Last Updated: 09/26/2026