Sami N. Malek

Sami N. Malek

Hematology | Oncology
1500 E Medical Center Dr
Floor B1 Reception A
Ann Arbor, MI 48109

Last Updated: 09/26/2026

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Found 52 publications

A plasma-based DNA test for quantification of disease burden in acute myeloid leukemia patients undergoing bone marrow transplantation.

A plasma-based DNA test for quantification of disease burden in acute myeloid leukemia patients undergoing bone marrow transplantation.

Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: April 14, 2026

A plasma-based DNA test for quantification of disease burden in acute myeloid leukemia patients undergoing bone marrow transplantation.

A plasma-based DNA test for quantification of disease burden in acute myeloid leukemia patients undergoing bone marrow transplantation.

Journal: medRxiv : the preprint server for health sciences
Published: February 23, 2026

Common variation at 1q23.3, 2p23.3, 2q33.3, and 2p21 influences risk of acute myeloid leukemia.

Common variation at 1q23.3, 2p23.3, 2q33.3, and 2p21 influences risk of acute myeloid leukemia.

Journal: Blood
Published: August 19, 2025

Common variation at 1q23.3, 2p23.3, 2q33.3, and 2p21 influences the risk of acute myeloid leukemia.

Common variation at 1q23.3, 2p23.3, 2q33.3, and 2p21 influences the risk of acute myeloid leukemia.

Journal: Blood
Published: August 19, 2025

Patients with AML with WT TP53 but defective TP53-mediated apoptosis have a dismal survival.

Patients with AML with WT TP53 but defective TP53-mediated apoptosis have a dismal survival.

Journal: JCI insight
Published: June 24, 2025

Understanding the Functional Dependence and Inhibition of the Bcl‑2 Pro-Survival Proteins in a Wide Spectrum of Cancers toward Precision Medicine.

Understanding the Functional Dependence and Inhibition of the Bcl‑2 Pro-Survival Proteins in a Wide Spectrum of Cancers toward Precision Medicine.

Journal: ACS pharmacology & translational science
Published: February 03, 2025

Correction: Aberrant BCAT1 expression augments MTOR activity and accelerates disease progression in chronic lymphocytic leukemia.

Correction: Aberrant BCAT1 expression augments MTOR activity and accelerates disease progression in chronic lymphocytic leukemia.

Journal: Leukemia
Published: November 13, 2024

Big1 is a newly identified autophagy regulator that is critical for a fully functional V-ATPase.

Big1 is a newly identified autophagy regulator that is critical for a fully functional V-ATPase.

Journal: Molecular biology of the cell
Published: September 11, 2024

STAT6 mutations compensate for CREBBP mutations and hyperactivate IL4/STAT6/RRAGD/mTOR signaling in follicular lymphoma.

STAT6 mutations compensate for CREBBP mutations and hyperactivate IL4/STAT6/RRAGD/mTOR signaling in follicular lymphoma.

Journal: Leukemia
Published: March 26, 2024

Aberrant BCAT1 expression augments MTOR activity and accelerates disease progression in chronic lymphocytic leukemia.

Aberrant BCAT1 expression augments MTOR activity and accelerates disease progression in chronic lymphocytic leukemia.

Journal: Leukemia
Published: January 30, 2024

Mutations in V-ATPase in follicular lymphoma activate autophagic flux creating a targetable dependency.

Mutations in V-ATPase in follicular lymphoma activate autophagic flux creating a targetable dependency.

Journal: Autophagy
Published: April 28, 2022

Follicular lymphoma-associated mutations in the V-ATPase chaperone VMA21 activate autophagy creating a targetable dependency.

Follicular lymphoma-associated mutations in the V-ATPase chaperone VMA21 activate autophagy creating a targetable dependency.

Journal: Autophagy
Published: March 15, 2022
Showing 1-12 of 52

Last Updated: 09/26/2026