Samuel Llopis-Pena

Heidelberg, BW 69120, Germany

Last Updated: 09/26/2026

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Found 38 publications

Correction: Raab et al. Truncated DAPK Variants Restore Tumor Suppressor Activity and Synergize with Standard Therapies in High-Grade Serous Ovarian Cancer. Cancers 2025, 17, 1910.

Correction: Raab et al. Truncated DAPK Variants Restore Tumor Suppressor Activity and Synergize with Standard Therapies in High-Grade Serous Ovarian Cancer. Cancers 2025, 17, 1910.

Journal: Cancers
Published: June 26, 2026

Autophagy suppression via SRC induction represents a therapeutic vulnerability for BAP1-mutant cancers.

Autophagy suppression via SRC induction represents a therapeutic vulnerability for BAP1-mutant cancers.

Journal: Autophagy
Published: August 04, 2025

Truncated DAPK Variants Restore Tumor Suppressor Activity and Synergize with Standard Therapies in High-Grade Serous Ovarian Cancer.

Truncated DAPK Variants Restore Tumor Suppressor Activity and Synergize with Standard Therapies in High-Grade Serous Ovarian Cancer.

Journal: Cancers
Published: April 30, 2025

Targeted reactivation of the novel tumor suppressor DAPK1, an upstream regulator of p53, in high-grade serous ovarian cancer by mRNA liposomes reduces viability and enhances drug sensitivity in preclinical models.

Targeted reactivation of the novel tumor suppressor DAPK1, an upstream regulator of p53, in high-grade serous ovarian cancer by mRNA liposomes reduces viability and enhances drug sensitivity in preclinical models.

Journal: Cancer communications (London, England)
Published: December 20, 2024

Synergistic Sensitization of High-Grade Serous Ovarian Cancer Cells Lacking Caspase-8 Expression to Chemotherapeutics Using Combinations of Small-Molecule BRD4 and CDK9 Inhibitors.

Synergistic Sensitization of High-Grade Serous Ovarian Cancer Cells Lacking Caspase-8 Expression to Chemotherapeutics Using Combinations of Small-Molecule BRD4 and CDK9 Inhibitors.

Journal: Cancers
Published: November 13, 2023

Rescue of p53 functions by in vitro-transcribed mRNA impedes the growth of high-grade serous ovarian cancer.

Rescue of p53 functions by in vitro-transcribed mRNA impedes the growth of high-grade serous ovarian cancer.

Journal: Cancer communications (London, England)
Published: July 31, 2023

GRB2 is a BECN1 interacting protein that regulates autophagy.

GRB2 is a BECN1 interacting protein that regulates autophagy.

Journal: Cell death & disease
Published: June 13, 2023

Correction: The non-apoptotic function of Caspase-8 in negatively regulating the CDK9-mediated Ser2 phosphorylation of RNA polymerase II in cervical cancer.

Correction: The non-apoptotic function of Caspase-8 in negatively regulating the CDK9-mediated Ser2 phosphorylation of RNA polymerase II in cervical cancer.

Journal: Cellular and molecular life sciences : CMLS
Published: January 05, 2023

The non-apoptotic function of Caspase-8 in negatively regulating the CDK9-mediated Ser2 phosphorylation of RNA polymerase II in cervical cancer.

The non-apoptotic function of Caspase-8 in negatively regulating the CDK9-mediated Ser2 phosphorylation of RNA polymerase II in cervical cancer.

Journal: Cellular and molecular life sciences : CMLS
Published: August 12, 2022

Nintedanib and Dasatinib Treatments Induce Protective Autophagy as a Potential Resistance Mechanism in MPM Cells.

Nintedanib and Dasatinib Treatments Induce Protective Autophagy as a Potential Resistance Mechanism in MPM Cells.

Journal: Frontiers in cell and developmental biology
Published: January 11, 2022

A Dual HiBiT-GFP-LC3 Lentiviral Reporter for Autophagy Flux Assessment.

A Dual HiBiT-GFP-LC3 Lentiviral Reporter for Autophagy Flux Assessment.

Journal: Methods in molecular biology (Clifton, N.J.)
Published: January 01, 2022

A new perspective on immune evasion: escaping immune surveillance by inactivating tumor suppressors.

A new perspective on immune evasion: escaping immune surveillance by inactivating tumor suppressors.

Journal: Signal transduction and targeted therapy
Published: October 20, 2021
Showing 1-12 of 38

Last Updated: 09/26/2026