Sandra C. Cardoso-Silva

Utrecht, UT, Netherlands

Last Updated: 09/26/2026

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Found 15 publications

Nuclear Receptor Subfamily 4A Signaling as a Key Disease Pathway of CD1c+ Dendritic Cell Dysregulation in Systemic Sclerosis.

Nuclear Receptor Subfamily 4A Signaling as a Key Disease Pathway of CD1c+ Dendritic Cell Dysregulation in Systemic Sclerosis.

Journal: Arthritis & rheumatology (Hoboken, N.J.)

Published: November 18, 2021

Suppression of IL-12/IL-23 p40 subunit in the skin and blood of psoriasis patients by Tofacitinib is dependent on active interferon-γ signaling in dendritic cells: Implications for the treatment of psoriasis and interferon-driven diseases.

Suppression of IL-12/IL-23 p40 subunit in the skin and blood of psoriasis patients by Tofacitinib is dependent on active interferon-γ signaling in dendritic cells: Implications for the treatment of psoriasis and interferon-driven diseases.

Journal: Experimental dermatology

Published: August 30, 2021

Hypoxia and TLR9 activation drive CXCL4 production in systemic sclerosis plasmacytoid dendritic cells via mtROS and HIF-2α.

Hypoxia and TLR9 activation drive CXCL4 production in systemic sclerosis plasmacytoid dendritic cells via mtROS and HIF-2α.

Journal: Rheumatology (Oxford, England)

Published: January 04, 2021

Implication of miR-126 and miR-139-5p in Plasmacytoid Dendritic Cell Dysregulation in Systemic Sclerosis.

Implication of miR-126 and miR-139-5p in Plasmacytoid Dendritic Cell Dysregulation in Systemic Sclerosis.

Journal: Journal of clinical medicine

Published: December 09, 2020

Longitudinal analysis of T-cell receptor repertoires reveals persistence of antigen-driven CD4+ and CD8+ T-cell clusters in systemic sclerosis.

Longitudinal analysis of T-cell receptor repertoires reveals persistence of antigen-driven CD4+ and CD8+ T-cell clusters in systemic sclerosis.

Journal: Journal of autoimmunity

Published: September 08, 2020

CXCL4 Links Inflammation and Fibrosis by Reprogramming Monocyte-Derived Dendritic Cells in vitro.

CXCL4 Links Inflammation and Fibrosis by Reprogramming Monocyte-Derived Dendritic Cells in vitro.

Journal: Frontiers in immunology

Published: April 24, 2020

CXCL4 suppresses tolerogenic immune signature of monocyte-derived dendritic cells.

CXCL4 suppresses tolerogenic immune signature of monocyte-derived dendritic cells.

Journal: European journal of immunology

Published: August 08, 2019

CXCL4 is a driver of cytokine mRNA stability in monocyte-derived dendritic cells.

CXCL4 is a driver of cytokine mRNA stability in monocyte-derived dendritic cells.

Journal: Molecular immunology

Published: May 25, 2019

Cytometry by time of flight identifies distinct signatures in patients with systemic sclerosis, systemic lupus erythematosus and Sjögrens syndrome.

Cytometry by time of flight identifies distinct signatures in patients with systemic sclerosis, systemic lupus erythematosus and Sjögrens syndrome.

Journal: European journal of immunology

Published: January 30, 2019

Histone modifications underlie monocyte dysregulation in patients with systemic sclerosis, underlining the treatment potential of epigenetic targeting.

Histone modifications underlie monocyte dysregulation in patients with systemic sclerosis, underlining the treatment potential of epigenetic targeting.

Journal: Annals of the rheumatic diseases

Published: August 16, 2018

Serum microRNA screening and functional studies reveal miR-483-5p as a potential driver of fibrosis in systemic sclerosis.

Serum microRNA screening and functional studies reveal miR-483-5p as a potential driver of fibrosis in systemic sclerosis.

Journal: Journal of autoimmunity

Published: October 09, 2017

CXCL4 is a novel inducer of human Th17 cells and correlates with IL-17 and IL-22 in psoriatic arthritis.

CXCL4 is a novel inducer of human Th17 cells and correlates with IL-17 and IL-22 in psoriatic arthritis.

Journal: European journal of immunology

Published: June 23, 2017

Showing 1-12 of 15

Last Updated: 09/26/2026