Sandra Larouche
Montreal, QC, Canada
Last Updated: 09/26/2026
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Found 16 publications
Grey matter degeneration during multiple sclerosis is linked to activation of neuronal necroptosis by oxidized phosphatidylcholines.
Journal: Research square
Published: July 10, 2026
Grey matter degeneration during multiple sclerosis is linked to activation of neuronal necroptosis by oxidized phosphatidylcholines.
Journal: Research square
Published: July 10, 2026
Age and Maturation Stage Linked Consequences of Fibrinogen on Human Oligodendroglia.
Journal: Journal of neuroscience research
Published: August 15, 2025
Oxidized phosphatidylcholines deposition drives chronic neurodegeneration in a mouse model of progressive multiple sclerosis via IL-1β signaling.
Journal: Nature neuroscience
Published: July 24, 2024
Single-Cell Transcriptomics Identifies Brain Endothelium Inflammatory Networks in Experimental Autoimmune Encephalomyelitis.
Journal: Neurology(R) neuroimmunology & neuroinflammation
Published: May 06, 2022
MCAM+ brain endothelial cells contribute to neuroinflammation by recruiting pathogenic CD4+ T lymphocytes.
Journal: Brain : a journal of neurology
Published: March 15, 2022
CLMP Promotes Leukocyte Migration Across Brain Barriers in Multiple Sclerosis.
Journal: Neurology(R) neuroimmunology & neuroinflammation
Published: February 23, 2022
DICAM promotes TH17 lymphocyte trafficking across the blood-brain barrier during autoimmune neuroinflammation.
Journal: Science translational medicine
Published: January 05, 2022
Validation of the French version of the Children's Alexithymia Measure.
Journal: L'Encephale
Published: May 22, 2020
Interleukin-26, preferentially produced by TH17 lymphocytes, regulates CNS barrier function.
Journal: Neurology(R) neuroimmunology & neuroinflammation
Published: April 18, 2020
Activated leukocyte cell adhesion molecule regulates B lymphocyte migration across central nervous system barriers.
Journal: Science translational medicine
Published: November 26, 2018
CD70 defines a subset of proinflammatory and CNS-pathogenic TH1/TH17 lymphocytes and is overexpressed in multiple sclerosis.
Journal: Cellular & molecular immunology
Published: October 21, 2018
Last Updated: 09/26/2026