Sandra M. Sacre
Brighton, ENG, United Kingdom
Last Updated: 09/26/2026
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Found 22 publications
Convalescent COVID-19 monocytes exhibit altered steady-state gene expression and reduced TLR2, TLR4 and RIG-I induced cytokine expression.
Journal: Human immunology
Published: August 23, 2024
Multiple TLRs elicit alternative NLRP3 inflammasome activation in primary human monocytes independent of RIPK1 kinase activity.
Journal: Frontiers in immunology
Published: November 08, 2022
The One That Got Away: How Macrophage-Derived IL-1β Escapes the Mycolactone-Dependent Sec61 Blockade in Buruli Ulcer.
Journal: Frontiers in immunology
Published: October 01, 2021
Contribution of Toll-Like Receptors and the NLRP3 Inflammasome in Rheumatoid Arthritis Pathophysiology.
Journal: ImmunoTargets and therapy
Published: May 06, 2021
Expression of sterile-α and armadillo motif containing protein (SARM) in rheumatoid arthritis monocytes correlates with TLR2-induced IL-1β and disease activity.
Journal: Rheumatology (Oxford, England)
Published: October 29, 2020
TLR expression profiles are a function of disease status in rheumatoid arthritis and experimental arthritis.
Journal: Journal of autoimmunity
Published: October 06, 2020
TLR1/2 and 5 induce elevated cytokine levels from rheumatoid arthritis monocytes independent of ACPA or RF autoantibody status.
Journal: Rheumatology (Oxford, England)
Published: January 20, 2020
Structural Modification of the Antidepressant Mianserin Suggests That Its Anti-inflammatory Activity May Be Independent of 5-Hydroxytryptamine Receptors.
Journal: Frontiers in immunology
Published: February 07, 2019
Precipitation of Soluble Uric Acid Is Necessary for In Vitro Activation of the NLRP3 Inflammasome in Primary Human Monocytes.
Journal: The Journal of rheumatology
Published: December 20, 2018
Differential induction of nuclear factor-like 2 signature genes with toll-like receptor stimulation.
Differential induction of nuclear factor-like 2 signature genes with toll-like receptor stimulation.
Journal: Free radical biology & medicine
Published: October 26, 2018
Engineering of TIMP-3 as a LAP-fusion protein for targeting to sites of inflammation.
Journal: Journal of cellular and molecular medicine
Published: August 03, 2018
Last Updated: 09/26/2026