Sandra Pavey

Woolloongabba, QLD, Australia

Last Updated: 09/26/2026

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Found 12 publications

Multiple interaction nodes define the postreplication repair response to UV-induced DNA damage that is defective in melanomas and correlated with UV signature mutation load.

Multiple interaction nodes define the postreplication repair response to UV-induced DNA damage that is defective in melanomas and correlated with UV signature mutation load.

Journal: Molecular oncology

Published: September 05, 2019

Short interfering RNA induced generation and translation of stable 5' mRNA cleavage intermediates.

Short interfering RNA induced generation and translation of stable 5' mRNA cleavage intermediates.

Journal: Biochimica et biophysica acta

Published: March 30, 2016

A novel ATM-dependent checkpoint defect distinct from loss of function mutation promotes genomic instability in melanoma.

A novel ATM-dependent checkpoint defect distinct from loss of function mutation promotes genomic instability in melanoma.

Journal: Pigment cell & melanoma research

Published: December 23, 2015

Multiparameter analysis of naevi and primary melanomas identifies a subset of naevi with elevated markers of transformation.

Multiparameter analysis of naevi and primary melanomas identifies a subset of naevi with elevated markers of transformation.

Journal: Pigment cell & melanoma research

Published: December 03, 2015

In vivo overexpression of Emi1 promotes chromosome instability and tumorigenesis.

In vivo overexpression of Emi1 promotes chromosome instability and tumorigenesis.

Journal: Oncogene

Published: April 30, 2015

Cyclin A/Cdk2 regulates Cdh1 and claspin during late S/G2 phase of the cell cycle.

Cyclin A/Cdk2 regulates Cdh1 and claspin during late S/G2 phase of the cell cycle.

Journal: Cell cycle (Georgetown, Tex.)

Published: December 09, 2014

Decatenation checkpoint-defective melanomas are dependent on PI3K for survival.

Decatenation checkpoint-defective melanomas are dependent on PI3K for survival.

Journal: Pigment cell & melanoma research

Published: February 10, 2014

DNA repair and cell cycle checkpoint defects as drivers and therapeutic targets in melanoma.

DNA repair and cell cycle checkpoint defects as drivers and therapeutic targets in melanoma.

Journal: Pigment cell & melanoma research

Published: June 03, 2013

Defective decatenation checkpoint function is a common feature of melanoma.

Defective decatenation checkpoint function is a common feature of melanoma.

Journal: The Journal of investigative dermatology

Published: December 05, 2012

A UVR-induced G2-phase checkpoint response to ssDNA gaps produced by replication fork bypass of unrepaired lesions is defective in melanoma.

A UVR-induced G2-phase checkpoint response to ssDNA gaps produced by replication fork bypass of unrepaired lesions is defective in melanoma.

Journal: The Journal of investigative dermatology

Published: March 10, 2012

A potent Chk1 inhibitor is selectively cytotoxic in melanomas with high levels of replicative stress.

A potent Chk1 inhibitor is selectively cytotoxic in melanomas with high levels of replicative stress.

Journal: Oncogene

Published: March 07, 2012

Defective cell cycle checkpoints as targets for anti-cancer therapies.

Defective cell cycle checkpoints as targets for anti-cancer therapies.

Journal: Frontiers in pharmacology

Published: October 17, 2011

Showing 1-12 of 12

Last Updated: 09/26/2026