Sara Lantigua
Santander, CB, Spain
Last Updated: 09/26/2026
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Found 8 publications
Prenatal, but not Postnatal, Curcumin Administration Rescues Neuromorphological and Cognitive Alterations in Ts65Dn Down Syndrome Mice.
Journal: The Journal of nutrition
Published: March 16, 2020
Prenatal Administration of Oleic Acid or Linolenic Acid Reduces Neuromorphological and Cognitive Alterations in Ts65dn Down Syndrome Mice.
Journal: The Journal of nutrition
Published: September 19, 2019
Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice.
Journal: Brain, behavior, and immunity
Published: December 12, 2017
Cerebellar alterations in a model of Down syndrome: The role of the Dyrk1A gene.
Journal: Neurobiology of disease
Published: September 19, 2017
Decreasing the Expression of GABAA α5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome.
Journal: Molecular neurobiology
Published: March 30, 2017
Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome.
Journal: Behavioural brain research
Published: March 11, 2017
Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes.
Journal: Neurobiology of disease
Published: December 10, 2016
Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome.
Journal: Neurochemical research
Published: May 30, 2016
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Last Updated: 09/26/2026