Sarah Mouche

Geneve, GE, Switzerland

Last Updated: 09/26/2026

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Found 14 publications

Non-genetic remodeling drives leukemia propagation and reveals actionable vulnerabilities in acute myeloid leukemia.

Non-genetic remodeling drives leukemia propagation and reveals actionable vulnerabilities in acute myeloid leukemia.

Journal: Cell reports. Medicine
Published: June 20, 2025

Targeting ferritinophagy impairs quiescent cancer stem cells in acute myeloid leukemia in vitro and in vivo models.

Targeting ferritinophagy impairs quiescent cancer stem cells in acute myeloid leukemia in vitro and in vivo models.

Journal: Science translational medicine
Published: July 24, 2024

The E3 ubiquitin ligase MARCH5 promotes mitochondrial fusion and cell-cycle progression in acute myeloid leukemia.

The E3 ubiquitin ligase MARCH5 promotes mitochondrial fusion and cell-cycle progression in acute myeloid leukemia.

Journal: Blood advances
Published: June 07, 2024

Very short insertions in the FLT3 gene are of therapeutic significance in acute myeloid leukemia.

Very short insertions in the FLT3 gene are of therapeutic significance in acute myeloid leukemia.

Journal: Blood advances
Published: October 12, 2023

Mitochondrial fusion is a therapeutic vulnerability of acute myeloid leukemia.

Mitochondrial fusion is a therapeutic vulnerability of acute myeloid leukemia.

Journal: Leukemia
Published: October 11, 2022

C/EBPα Confers Dependence to Fatty Acid Anabolic Pathways and Vulnerability to Lipid Oxidative Stress-Induced Ferroptosis in FLT3-Mutant Leukemia.

C/EBPα Confers Dependence to Fatty Acid Anabolic Pathways and Vulnerability to Lipid Oxidative Stress-Induced Ferroptosis in FLT3-Mutant Leukemia.

Journal: Cancer discovery
Published: April 15, 2022

RAS activation induces synthetic lethality of MEK inhibition with mitochondrial oxidative metabolism in acute myeloid leukemia.

RAS activation induces synthetic lethality of MEK inhibition with mitochondrial oxidative metabolism in acute myeloid leukemia.

Journal: Leukemia
Published: December 07, 2021

AMP-Activated Protein Kinase Contributes to Apoptosis Induced by the Bcl-2 Inhibitor Venetoclax in Acute Myeloid Leukemia.

AMP-Activated Protein Kinase Contributes to Apoptosis Induced by the Bcl-2 Inhibitor Venetoclax in Acute Myeloid Leukemia.

Journal: Cancers
Published: September 14, 2021

AMPK-PERK axis represses oxidative metabolism and enhances apoptotic priming of mitochondria in acute myeloid leukemia.

AMPK-PERK axis represses oxidative metabolism and enhances apoptotic priming of mitochondria in acute myeloid leukemia.

Journal: Cell reports
Published: June 15, 2021

An In Vivo CRISPR Screening Platform for Prioritizing Therapeutic Targets in AML.

An In Vivo CRISPR Screening Platform for Prioritizing Therapeutic Targets in AML.

Journal: Cancer discovery
Published: December 23, 2020

Adrenomedullin-CALCRL axis controls relapse-initiating drug tolerant acute myeloid leukemia cells.

Adrenomedullin-CALCRL axis controls relapse-initiating drug tolerant acute myeloid leukemia cells.

Journal: Nature communications
Published: August 11, 2020

Beta-Cell-Specific Expression of Nicotinamide Adenine Dinucleotide Phosphate Oxidase 5 Aggravates High-Fat Diet-Induced Impairment of Islet Insulin Secretion in Mice.

Beta-Cell-Specific Expression of Nicotinamide Adenine Dinucleotide Phosphate Oxidase 5 Aggravates High-Fat Diet-Induced Impairment of Islet Insulin Secretion in Mice.

Journal: Antioxidants & redox signaling
Published: January 15, 2020
Showing 1-12 of 14

Last Updated: 09/26/2026