Sarka Lhotak

Hamilton, ON, Canada

Last Updated: 09/26/2026

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Found 24 publications

Correction: Autoantibodies against the cell surface-associated chaperone GRP78 stimulate tumor growth via tissue factor.

Correction: Autoantibodies against the cell surface-associated chaperone GRP78 stimulate tumor growth via tissue factor.

Journal: The Journal of biological chemistry

Published: August 15, 2025

Restoration of the ER stress response protein TDAG51 in hepatocytes mitigates NAFLD in mice.

Restoration of the ER stress response protein TDAG51 in hepatocytes mitigates NAFLD in mice.

Journal: The Journal of biological chemistry

Published: October 24, 2023

Salsalate reduces atherosclerosis through AMPKβ1 in mice.

Salsalate reduces atherosclerosis through AMPKβ1 in mice.

Journal: Molecular metabolism

Published: July 04, 2021

TDAG51 (T-Cell Death-Associated Gene 51) Is a Key Modulator of Vascular Calcification and Osteogenic Transdifferentiation of Arterial Smooth Muscle Cells.

TDAG51 (T-Cell Death-Associated Gene 51) Is a Key Modulator of Vascular Calcification and Osteogenic Transdifferentiation of Arterial Smooth Muscle Cells.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: May 22, 2020

Pcsk9 knockout exacerbates diet-induced non-alcoholic steatohepatitis, fibrosis and liver injury in mice.

Pcsk9 knockout exacerbates diet-induced non-alcoholic steatohepatitis, fibrosis and liver injury in mice.

Journal: JHEP reports : innovation in hepatology

Published: April 13, 2019

Sialidase down-regulation reduces non-HDL cholesterol, inhibits leukocyte transmigration, and attenuates atherosclerosis in ApoE knockout mice.

Sialidase down-regulation reduces non-HDL cholesterol, inhibits leukocyte transmigration, and attenuates atherosclerosis in ApoE knockout mice.

Journal: The Journal of biological chemistry

Published: June 26, 2018

Anti-GRP78 autoantibodies induce endothelial cell activation and accelerate the development of atherosclerotic lesions.

Anti-GRP78 autoantibodies induce endothelial cell activation and accelerate the development of atherosclerotic lesions.

Journal: JCI insight

Published: December 19, 2017

Loss-of-function PCSK9 mutants evade the unfolded protein response sensor GRP78 and fail to induce endoplasmic reticulum stress when retained.

Loss-of-function PCSK9 mutants evade the unfolded protein response sensor GRP78 and fail to induce endoplasmic reticulum stress when retained.

Journal: The Journal of biological chemistry

Published: November 22, 2017

IL-6 mediates ER expansion during hyperpolarization of alternatively activated macrophages.

IL-6 mediates ER expansion during hyperpolarization of alternatively activated macrophages.

Journal: Immunology and cell biology

Published: November 21, 2017

Autoantibodies against the cell surface-associated chaperone GRP78 stimulate tumor growth via tissue factor.

Autoantibodies against the cell surface-associated chaperone GRP78 stimulate tumor growth via tissue factor.

Journal: The Journal of biological chemistry

Published: May 31, 2017

Characterization of Proliferating Lesion-Resident Cells During All Stages of Atherosclerotic Growth.

Characterization of Proliferating Lesion-Resident Cells During All Stages of Atherosclerotic Growth.

Journal: Journal of the American Heart Association

Published: August 17, 2016

Endoplasmic Reticulum Stress and Ca2+ Depletion Differentially Modulate the Sterol Regulatory Protein PCSK9 to Control Lipid Metabolism.

Endoplasmic Reticulum Stress and Ca2+ Depletion Differentially Modulate the Sterol Regulatory Protein PCSK9 to Control Lipid Metabolism.

Journal: The Journal of biological chemistry

Published: June 17, 2016

Showing 1-12 of 24

Last Updated: 09/26/2026