Schafiq Nabhani
Duesseldorf, NW, Germany
Last Updated: 09/26/2026
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Found 12 publications
Unveiling the Mechanism: Injectable Poly-L-Lactic Acid's Evolving Role-Insights From Recent Studies.
Unveiling the Mechanism: Injectable Poly-L-Lactic Acid's Evolving Role-Insights From Recent Studies.
Journal: Journal of cosmetic dermatology
Published: May 24, 2024
Activity of amorolfine or ciclopirox in combination with terbinafine against pathogenic fungi in onychomycosis-Results of an in vitro investigation.
Journal: Mycoses
Published: December 20, 2023
Prostaglandin receptor EP4 expression by Th17 cells is associated with high disease activity in ankylosing spondylitis.
Journal: Arthritis research & therapy
Published: December 12, 2018
EBV Negative Lymphoma and Autoimmune Lymphoproliferative Syndrome Like Phenotype Extend the Clinical Spectrum of Primary Immunodeficiency Caused by STK4 Deficiency.
Journal: Frontiers in immunology
Published: June 29, 2018
STAT3 gain-of-function mutations associated with autoimmune lymphoproliferative syndrome like disease deregulate lymphocyte apoptosis and can be targeted by BH3 mimetic compounds.
Journal: Clinical immunology (Orlando, Fla.)
Published: September 28, 2016
Specific antibody deficiency and autoinflammatory disease extend the clinical and immunological spectrum of heterozygous NFKB1 loss-of-function mutations in humans.
Journal: Haematologica
Published: July 02, 2016
De novo PIK3R1 gain-of-function with recurrent sinopulmonary infections, long-lasting chronic CMV-lymphadenitis and microcephaly.
Journal: Clinical immunology (Orlando, Fla.)
Published: August 21, 2015
Early-onset Evans syndrome, immunodeficiency, and premature immunosenescence associated with tripeptidyl-peptidase II deficiency.
Journal: Blood
Published: November 22, 2014
Autoimmune lymphoproliferative syndrome-like disease in patients with LRBA mutation.
Journal: Clinical immunology (Orlando, Fla.)
Published: November 10, 2014
A novel homozygous Fas ligand mutation leads to early protein truncation, abrogation of death receptor and reverse signaling and a severe form of the autoimmune lymphoproliferative syndrome.
Journal: Clinical immunology (Orlando, Fla.)
Published: October 07, 2014
The cell cycle regulator CCDC6 is a key target of RNA-binding protein EWS.
Journal: PloS one
Published: May 08, 2014
Showing 1-12 of 12
Last Updated: 09/26/2026