Sebastian Honnen

Duesseldorf, NW 40225, Germany

Last Updated: 09/26/2026

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Found 13 publications

RNA is a pro-apoptotic target of cisplatin in cancer cell lines and C. elegans.

RNA is a pro-apoptotic target of cisplatin in cancer cell lines and C. elegans.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
Published: February 05, 2024

Cisplatin-induced neurotoxicity involves the disruption of serotonergic neurotransmission.

Cisplatin-induced neurotoxicity involves the disruption of serotonergic neurotransmission.

Journal: Pharmacological research
Published: June 30, 2021

Insights into cisplatin-induced neurotoxicity and mitochondrial dysfunction in Caenorhabditis elegans.

Insights into cisplatin-induced neurotoxicity and mitochondrial dysfunction in Caenorhabditis elegans.

Journal: Disease models & mechanisms
Published: June 11, 2021

Use of C. elegans as a 3R-compliant in vivo model for the chemoprevention of cisplatin-induced neurotoxicity.

Use of C. elegans as a 3R-compliant in vivo model for the chemoprevention of cisplatin-induced neurotoxicity.

Journal: Experimental neurology
Published: December 11, 2020

The mycotoxin beauvericin impairs development, fertility and life span in the nematode Caenorhabditis elegans accompanied by increased germ cell apoptosis and lipofuscin accumulation.

The mycotoxin beauvericin impairs development, fertility and life span in the nematode Caenorhabditis elegans accompanied by increased germ cell apoptosis and lipofuscin accumulation.

Journal: Toxicology letters
Published: April 09, 2020

Statins Induce a DAF-16/Foxo-dependent Longevity Phenotype via JNK-1 through Mevalonate Depletion in C. elegans.

Statins Induce a DAF-16/Foxo-dependent Longevity Phenotype via JNK-1 through Mevalonate Depletion in C. elegans.

Journal: Aging and disease
Published: December 03, 2018

BRCA1 and BARD1 mediate apoptotic resistance but not longevity upon mitochondrial stress in Caenorhabditis elegans.

BRCA1 and BARD1 mediate apoptotic resistance but not longevity upon mitochondrial stress in Caenorhabditis elegans.

Journal: EMBO reports
Published: January 24, 2018

Genetic and cellular sensitivity of Caenorhabditis elegans to the chemotherapeutic agent cisplatin.

Genetic and cellular sensitivity of Caenorhabditis elegans to the chemotherapeutic agent cisplatin.

Journal: Disease models & mechanisms
Published: January 08, 2018

Multiple DNA damage-dependent and DNA damage-independent stress responses define the outcome of ATR/Chk1 targeting in medulloblastoma cells.

Multiple DNA damage-dependent and DNA damage-independent stress responses define the outcome of ATR/Chk1 targeting in medulloblastoma cells.

Journal: Cancer letters
Published: December 21, 2017

Caenorhabditis elegans as a powerful alternative model organism to promote research in genetic toxicology and biomedicine.

Caenorhabditis elegans as a powerful alternative model organism to promote research in genetic toxicology and biomedicine.

Journal: Archives of toxicology
Published: November 15, 2016

Methylated derivatives of myricetin enhance life span in Caenorhabditis elegans dependent on the transcription factor DAF-16.

Methylated derivatives of myricetin enhance life span in Caenorhabditis elegans dependent on the transcription factor DAF-16.

Journal: Food & function
Published: August 19, 2015

TSG (2,3,5,4'-Tetrahydroxystilbene-2-O- β -D-glucoside) from the Chinese Herb Polygonum multiflorum Increases Life Span and Stress Resistance of Caenorhabditis elegans.

TSG (2,3,5,4'-Tetrahydroxystilbene-2-O- β -D-glucoside) from the Chinese Herb Polygonum multiflorum Increases Life Span and Stress Resistance of Caenorhabditis elegans.

Journal: Oxidative medicine and cellular longevity
Published: June 13, 2014
Showing 1-12 of 13

Last Updated: 09/26/2026