Selma Myers-Osmanagic
Währinger Str. 10
Vienna, 1090, Austria
Vienna, 1090, Austria
Last Updated: 09/26/2026
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Found 14 publications
The unfolded protein response in progeria arteries originates from non-endothelial cell types.
Journal: Life science alliance
Published: August 13, 2025
Plecstatin inhibits hepatocellular carcinoma tumorigenesis and invasion through cytolinker plectin.
Journal: Molecular oncology
Published: August 11, 2025
Hepatic Lipoprotein Metabolism: Current and Future In Vitro Cell-Based Systems.
Journal: Biomolecules
Published: May 14, 2025
Premature aging in genetic diseases: what conclusions can be drawn for physiological aging.
Journal: Frontiers in aging
Published: October 25, 2023
Aging impairs the neurovascular interface in the heart.
Journal: Science (New York, N.Y.)
Published: August 24, 2023
Endothelial and systemic upregulation of miR-34a-5p fine-tunes senescence in progeria.
Journal: Aging
Published: September 25, 2021
The structural and gene expression hypotheses in laminopathic diseases-not so different after all.
Journal: Molecular biology of the cell
Published: July 16, 2019
Endothelial progerin expression causes cardiovascular pathology through an impaired mechanoresponse.
Endothelial progerin expression causes cardiovascular pathology through an impaired mechanoresponse.
Journal: The Journal of clinical investigation
Published: March 27, 2018
Arteriovenous specification: BMPER and TWSG1 determine endothelial cell fate via activation of synergistic BMP and Notch signaling.
Journal: The FEBS journal
Published: March 12, 2018
Plectin reinforces vascular integrity by mediating crosstalk between the vimentin and the actin networks.
Journal: Journal of cell science
Published: March 24, 2015
Last Updated: 09/26/2026