Seong O. Park
Iksan, Republic of Korea
Last Updated: 09/26/2026
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Found 16 publications
Myeloid miR-155 deficiency exacerbates viral encephalitis by hindering M1 macrophage polarization due to impaired NLRP3 inflammasome activation in extraneural tissues.
Journal: Frontiers in immunology
Published: February 26, 2026
The gut microbiota limits systemic inflammation during neurotrophic viral CNS infection by priming tonic type I interferon signaling.
Journal: Journal of neuroinflammation
Published: August 21, 2025
Myeloid cAMP Reduction Shifts Rhinovirus-Induced Airway Inflammation From Neutrophilic to Eosinophilic by Suppressing M1-Interstitial Macrophages.
Journal: Allergy
Published: September 27, 2024
Inhibition of NADPH oxidase 2 enhances resistance to viral neuroinflammation by facilitating M1-polarization of macrophages at the extraneural tissues.
Journal: Journal of neuroinflammation
Published: October 11, 2023
TLR3/TRIF pathway confers protection against herpes simplex encephalitis through NK cell activation mediated by a loop of type I IFN and IL-15 from epithelial and dendritic cells.
Journal: Immunology
Published: November 26, 2022
Influence of body fatness on propofol requirements for loss of consciousness in target-controlled infusion: A STROBE-compliant study.
Journal: Medicine
Published: September 15, 2022
T cell-intrinsic miR-155 is required for Th2 and Th17-biased responses in acute and chronic airway inflammation by targeting several different transcription factors.
Journal: Immunology
Published: October 30, 2021
Indoleamine 2,3-Dioxygenase in Hematopoietic Stem Cell-Derived Cells Suppresses Rhinovirus-Induced Neutrophilic Airway Inflammation by Regulating Th1- and Th17-Type Responses.
Journal: Immune network
Published: June 25, 2021
Type I IFN signaling limits hemorrhage-like disease after infection with Japanese encephalitis virus through modulating a prerequisite infection of CD11b+Ly-6C+ monocytes.
Journal: Journal of neuroinflammation
Published: December 04, 2020
Last Updated: 09/26/2026