Sevasti Flytzani
Stockholm, AB, Sweden
Last Updated: 09/26/2026
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Found 8 publications
C-type lectin receptors Mcl and Mincle control development of multiple sclerosis-like neuroinflammation.
Journal: The Journal of clinical investigation
Published: October 29, 2018
Translational utility of experimental autoimmune encephalomyelitis: recent developments.
Journal: Journal of inflammation research
Published: December 02, 2015
MOG-induced experimental autoimmune encephalomyelitis in the rat species triggers anti-neurofascin antibody response that is genetically regulated.
Journal: Journal of neuroinflammation
Published: May 25, 2015
Rat bone marrow-derived dendritic cells generated with GM-CSF/IL-4 or FLT3L exhibit distinct phenotypical and functional characteristics.
Journal: Journal of leukocyte biology
Published: September 10, 2014
Combining genetic mapping with genome-wide expression in experimental autoimmune encephalomyelitis highlights a gene network enriched for T cell functions and candidate genes regulating autoimmunity.
Journal: Human molecular genetics
Published: August 01, 2013
Parent-of-origin effects implicate epigenetic regulation of experimental autoimmune encephalomyelitis and identify imprinted Dlk1 as a novel risk gene.
Journal: PLoS genetics
Published: July 29, 2013
Anti-MOG antibodies are under polygenic regulation with the most significant control coming from the C-type lectin-like gene locus.
Journal: Genes and immunity
Published: March 25, 2013
Genetic variability in the rat Aplec C-type lectin gene cluster regulates lymphocyte trafficking and motor neuron survival after traumatic nerve root injury.
Journal: Journal of neuroinflammation
Published: January 14, 2013
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Last Updated: 09/26/2026