Shaojie G. Ding

1 Xueshi Road
Hangzhou, 310006, China

Last Updated: 09/26/2026

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Found 24 publications

Immune escape in endometriosis: mechanisms, reprogramming strategies, and precision interventions.

Immune escape in endometriosis: mechanisms, reprogramming strategies, and precision interventions.

Journal: Reproductive biology and endocrinology : RB&E

Published: February 04, 2026

LINC00632 combines with ERα to target VISTA expression, ameliorating endometriosis progression.

LINC00632 combines with ERα to target VISTA expression, ameliorating endometriosis progression.

Journal: iScience

Published: May 30, 2025

Bisphenol analogues in soils and lettuce (Lactuca sativa L.) around typical factories in eastern China: Occurrence, contamination characteristics, and health risks.

Bisphenol analogues in soils and lettuce (Lactuca sativa L.) around typical factories in eastern China: Occurrence, contamination characteristics, and health risks.

Journal: Environmental pollution (Barking, Essex : 1987)

Published: November 26, 2024

CCL2/CCR4 participates in mast cell-mediated epithelial mesenchymal transition in endometriosis.

CCL2/CCR4 participates in mast cell-mediated epithelial mesenchymal transition in endometriosis.

Journal: Reproductive biomedicine online

Published: June 12, 2024

CCL18 promotes endometriosis by increasing endometrial cell migration and neuroangiogenesis.

CCL18 promotes endometriosis by increasing endometrial cell migration and neuroangiogenesis.

Journal: European journal of histochemistry : EJH

Published: April 22, 2024

GPR30-mediated non-classic estrogen pathway in mast cells participates in endometriosis pain via the production of FGF2.

GPR30-mediated non-classic estrogen pathway in mast cells participates in endometriosis pain via the production of FGF2.

Journal: Frontiers in immunology

Published: November 24, 2022

Anemia-Associated Platelets and Plasma Prothrombin Time Increase in Patients with Adenomyosis.

Anemia-Associated Platelets and Plasma Prothrombin Time Increase in Patients with Adenomyosis.

Journal: Journal of clinical medicine

Published: June 19, 2022

NLRP3 Inflammasome Activation of Mast Cells by Estrogen via the Nuclear-Initiated Signaling Pathway Contributes to the Development of Endometriosis.

NLRP3 Inflammasome Activation of Mast Cells by Estrogen via the Nuclear-Initiated Signaling Pathway Contributes to the Development of Endometriosis.

Journal: Frontiers in immunology

Published: July 30, 2021

RON Mediates Tumor-Promoting Effects in Endometrial Adenocarcinoma.

RON Mediates Tumor-Promoting Effects in Endometrial Adenocarcinoma.

Journal: BioMed research international

Published: April 13, 2021

Macrophage-derived netrin-1 contributes to endometriosis-associated pain.

Macrophage-derived netrin-1 contributes to endometriosis-associated pain.

Journal: Annals of translational medicine

Published: February 08, 2021

Recepteur d'origine nantais contributes to the development of endometriosis via promoting epithelial-mesenchymal transition of a endometrial epithelial cells.

Recepteur d'origine nantais contributes to the development of endometriosis via promoting epithelial-mesenchymal transition of a endometrial epithelial cells.

Journal: Journal of cellular and molecular medicine

Published: August 07, 2020

Possible involvement of crosstalk between endometrial cells and mast cells in the development of endometriosis via CCL8/CCR1.

Possible involvement of crosstalk between endometrial cells and mast cells in the development of endometriosis via CCL8/CCR1.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: March 25, 2020

Showing 1-12 of 24

Last Updated: 09/26/2026