Shaoqun Shu

Changsha, China

Last Updated: 09/26/2026

Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 19 publications

Establishment and characterization of a mouse model for studying kidney repair in diabetes.

Establishment and characterization of a mouse model for studying kidney repair in diabetes.

Journal: American journal of physiology. Renal physiology
Published: September 12, 2024

Tubular cells produce FGF2 via autophagy after acute kidney injury leading to fibroblast activation and renal fibrosis.

Tubular cells produce FGF2 via autophagy after acute kidney injury leading to fibroblast activation and renal fibrosis.

Journal: Autophagy
Published: May 02, 2022

Endoplasmic reticulum stress contributes to cisplatin-induced chronic kidney disease via the PERK-PKCδ pathway.

Endoplasmic reticulum stress contributes to cisplatin-induced chronic kidney disease via the PERK-PKCδ pathway.

Journal: Cellular and molecular life sciences : CMLS
Published: April 02, 2022

Reciprocal regulation between ER stress and autophagy in renal tubular fibrosis and apoptosis.

Reciprocal regulation between ER stress and autophagy in renal tubular fibrosis and apoptosis.

Journal: Cell death & disease
Published: April 11, 2021

NAM protects against cisplatin-induced acute kidney injury by suppressing the PARP1/p53 pathway.

NAM protects against cisplatin-induced acute kidney injury by suppressing the PARP1/p53 pathway.

Journal: Toxicology and applied pharmacology
Published: January 13, 2021

Corrigendum to "Endoplasmic reticulum stress is activated in post-ischemic kidneys to promote chronic kidney disease" [EBioMedicine 37 (2018) 269-280].

Corrigendum to "Endoplasmic reticulum stress is activated in post-ischemic kidneys to promote chronic kidney disease" [EBioMedicine 37 (2018) 269-280].

Journal: EBioMedicine
Published: January 08, 2021

AMPK/mTOR Signaling in Autophagy Regulation During Cisplatin-Induced Acute Kidney Injury.

AMPK/mTOR Signaling in Autophagy Regulation During Cisplatin-Induced Acute Kidney Injury.

Journal: Frontiers in physiology
Published: October 21, 2020

The negative feedback loop of NF-κB/miR-376b/NFKBIZ in septic acute kidney injury.

The negative feedback loop of NF-κB/miR-376b/NFKBIZ in septic acute kidney injury.

Journal: JCI insight
Published: July 14, 2020

The PINK1/PARK2/optineurin pathway of mitophagy is activated for protection in septic acute kidney injury.

The PINK1/PARK2/optineurin pathway of mitophagy is activated for protection in septic acute kidney injury.

Journal: Redox biology
Published: July 11, 2020

Red Blood Cell Distribution Width and Neutrophil-to-Lymphocyte Ratio in Predicting Adverse Outcomes of Acute Kidney Injury in Hospitalized Patients.

Red Blood Cell Distribution Width and Neutrophil-to-Lymphocyte Ratio in Predicting Adverse Outcomes of Acute Kidney Injury in Hospitalized Patients.

Journal: Kidney diseases (Basel, Switzerland)
Published: February 08, 2020

Irisin is induced in renal ischemia-reperfusion to protect against tubular cell injury via suppressing p53.

Irisin is induced in renal ischemia-reperfusion to protect against tubular cell injury via suppressing p53.

Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: January 23, 2020

Protein Kinase C-δ Mediates Kidney Tubular Injury in Cold Storage-Associated Kidney Transplantation.

Protein Kinase C-δ Mediates Kidney Tubular Injury in Cold Storage-Associated Kidney Transplantation.

Journal: Journal of the American Society of Nephrology : JASN
Published: October 15, 2019
Showing 1-12 of 19

Last Updated: 09/26/2026