Shashikanta Sahoo

Ghaziabad, UP, India

Last Updated: 09/26/2026

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Found 10 publications

Doxorubicin Resistance Reprograms Triple-Negative Breast Cancer Cell Metabolism via the Fatty Acid β-Oxidation (FAO)-CD36 Regulatory Circuit: Relevance of Enhanced FAO on Tumor Cell Invasiveness.

Doxorubicin Resistance Reprograms Triple-Negative Breast Cancer Cell Metabolism via the Fatty Acid β-Oxidation (FAO)-CD36 Regulatory Circuit: Relevance of Enhanced FAO on Tumor Cell Invasiveness.

Journal: Molecular carcinogenesis

Published: October 04, 2025

Metadherin with Stromal-Immune Cues Drives CD36-Dependent Lipid Reprogramming and Metastasis in Triple-Negative Breast Cancer: Insights from a Hetero-Spheroid Model.

Metadherin with Stromal-Immune Cues Drives CD36-Dependent Lipid Reprogramming and Metastasis in Triple-Negative Breast Cancer: Insights from a Hetero-Spheroid Model.

Journal: Advanced healthcare materials

Published: October 02, 2025

Palmitic acid fuels triple-negative breast cancer through metadherin-dependent fatty acid β-oxidation: Relevance to high fat diet-induced breast cancer progression.

Palmitic acid fuels triple-negative breast cancer through metadherin-dependent fatty acid β-oxidation: Relevance to high fat diet-induced breast cancer progression.

Journal: Cellular signalling

Published: July 23, 2025

MTDH∆7-mediated mTOR activation drives doxorubicin resistance in triple-negative breast cancer: Relevance of mTORC1 inhibition on chemosensitization.

MTDH∆7-mediated mTOR activation drives doxorubicin resistance in triple-negative breast cancer: Relevance of mTORC1 inhibition on chemosensitization.

Journal: Cellular signalling

Published: December 20, 2024

SIRT1 promotes doxorubicin-induced breast cancer drug resistance and tumor angiogenesis via regulating GSH-mediated redox homeostasis.

SIRT1 promotes doxorubicin-induced breast cancer drug resistance and tumor angiogenesis via regulating GSH-mediated redox homeostasis.

Journal: Molecular carcinogenesis

Published: April 30, 2024

Mitochondria-targeted esculetin and metformin delay endothelial senescence by promoting fatty acid β-oxidation: Relevance in age-associated atherosclerosis.

Mitochondria-targeted esculetin and metformin delay endothelial senescence by promoting fatty acid β-oxidation: Relevance in age-associated atherosclerosis.

Journal: Mechanisms of ageing and development

Published: January 05, 2024

Correction to: Therapeutic efficacies of mitochondria‑targeted esculetin and metformin in the improvement of age‑associated atherosclerosis via regulating AMPK activation.

Correction to: Therapeutic efficacies of mitochondria‑targeted esculetin and metformin in the improvement of age‑associated atherosclerosis via regulating AMPK activation.

Journal: GeroScience

Published: December 25, 2023

Mitochondria targeted esculetin administration improves insulin resistance and hyperglycemia-induced atherosclerosis in db/db mice.

Mitochondria targeted esculetin administration improves insulin resistance and hyperglycemia-induced atherosclerosis in db/db mice.

Journal: Journal of molecular medicine (Berlin, Germany)

Published: November 14, 2023

Therapeutic efficacies of mitochondria-targeted esculetin and metformin in the improvement of age-associated atherosclerosis via regulating AMPK activation.

Therapeutic efficacies of mitochondria-targeted esculetin and metformin in the improvement of age-associated atherosclerosis via regulating AMPK activation.

Journal: GeroScience

Published: June 23, 2023

DOT1L regulates MTDH-mediated angiogenesis in triple-negative breast cancer: intermediacy of NF-κB-HIF1α axis.

DOT1L regulates MTDH-mediated angiogenesis in triple-negative breast cancer: intermediacy of NF-κB-HIF1α axis.

Journal: The FEBS journal

Published: April 06, 2022

Showing 1-10 of 10

Last Updated: 09/26/2026