Shenghui Li

Tianjin, 300052, China

Last Updated: 09/26/2026

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Found 22 publications

A Programmed Streptavidin-Condensed Bifunctional Nucleic Acid Nanoplatform Breaking the Vicious Cycle for Traumatic Brain Injury Therapy.

A Programmed Streptavidin-Condensed Bifunctional Nucleic Acid Nanoplatform Breaking the Vicious Cycle for Traumatic Brain Injury Therapy.

Journal: ACS nano
Published: May 01, 2026

Distance-Independent Synergistic Fluorescence Quenching Lateral Flow Immunoassay for Sensitive Detection of Creatine Kinase-MB Based on Gold-Iridium Oxide Nanoparticles.

Distance-Independent Synergistic Fluorescence Quenching Lateral Flow Immunoassay for Sensitive Detection of Creatine Kinase-MB Based on Gold-Iridium Oxide Nanoparticles.

Journal: ACS sensors
Published: November 10, 2025

Simply Engineered crRNA with CRISPR/Cas12a System Enables Wide-Scope Nucleic Acid Biomarker Analysis.

Simply Engineered crRNA with CRISPR/Cas12a System Enables Wide-Scope Nucleic Acid Biomarker Analysis.

Journal: Nano letters
Published: May 12, 2025

Neutrophil extracellular traps induce endothelial damage and exacerbate vasospasm in traumatic brain injury.

Neutrophil extracellular traps induce endothelial damage and exacerbate vasospasm in traumatic brain injury.

Journal: Theranostics
Published: April 14, 2025

Corrigendum to "Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury" [International Immunopharmacology 130 (2024) 111674].

Corrigendum to "Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury" [International Immunopharmacology 130 (2024) 111674].

Journal: International immunopharmacology
Published: March 27, 2025

Neutrophil extracellular traps aggravate neuronal apoptosis and neuroinflammation via neddylation after traumatic brain injury.

Neutrophil extracellular traps aggravate neuronal apoptosis and neuroinflammation via neddylation after traumatic brain injury.

Journal: Theranostics
Published: February 04, 2025

IL-23 promotes neuronal ferroptosis via IL-23R/STAT3 signaling after traumatic brain injury.

IL-23 promotes neuronal ferroptosis via IL-23R/STAT3 signaling after traumatic brain injury.

Journal: Cell communication and signaling : CCS
Published: January 26, 2025

Inhibition of S100A8/A9 ameliorates neuroinflammation by blocking NET formation following traumatic brain injury.

Inhibition of S100A8/A9 ameliorates neuroinflammation by blocking NET formation following traumatic brain injury.

Journal: Redox biology
Published: December 13, 2024

Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury.

Brain-derived extracellular vesicles mediate systemic coagulopathy and inflammation after traumatic brain injury.

Journal: International immunopharmacology
Published: November 27, 2023

TIMP1/CHI3L1 facilitates glioma progression and immunosuppression via NF-κB activation.

TIMP1/CHI3L1 facilitates glioma progression and immunosuppression via NF-κB activation.

Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: September 18, 2023

Inactivation of ERK1/2 signaling mediates dysfunction of basal meningeal lymphatic vessels in experimental subdural hematoma.

Inactivation of ERK1/2 signaling mediates dysfunction of basal meningeal lymphatic vessels in experimental subdural hematoma.

Journal: Theranostics
Published: June 30, 2023

Inhibition of neutrophil extracellular trap formation ameliorates neuroinflammation and neuronal apoptosis via STING-dependent IRE1α/ASK1/JNK signaling pathway in mice with traumatic brain injury.

Inhibition of neutrophil extracellular trap formation ameliorates neuroinflammation and neuronal apoptosis via STING-dependent IRE1α/ASK1/JNK signaling pathway in mice with traumatic brain injury.

Journal: Journal of neuroinflammation
Published: April 19, 2023
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Last Updated: 09/26/2026