Shengwei Zhang
Wuhan, China
Last Updated: 09/26/2026
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Found 9 publications
Vimentin inhibits α-tubulin acetylation via enhancing α-TAT1 degradation to suppress the replication of human parainfluenza virus type 3.
Journal: PLoS pathogens
Published: June 10, 2022
Involvement of Actin-Regulating Factor Cofilin in the Inclusion Body Formation and RNA Synthesis of Human Parainfluenza Virus Type 3 via Interaction With the Nucleoprotein.
Journal: Frontiers in microbiology
Published: October 31, 2018
An alanine residue in human parainfluenza virus type 3 phosphoprotein is critical for restricting excessive N0-P interaction and maintaining N solubility.
Journal: Virology
Published: October 10, 2017
A leucine residue in the C terminus of human parainfluenza virus type 3 matrix protein is essential for efficient virus-like particle and virion release.
Journal: Journal of virology
Published: September 05, 2014
Several residues within the N-terminal arm of vesicular stomatitis virus nucleoprotein play a critical role in protecting viral RNA from nuclease digestion.
Journal: Virology
Published: July 11, 2014
An amino acid of human parainfluenza virus type 3 nucleoprotein is critical for template function and cytoplasmic inclusion body formation.
Journal: Journal of virology
Published: September 13, 2013
N-terminal phosphorylation of phosphoprotein of vesicular stomatitis virus is required for preventing nucleoprotein from binding to cellular RNAs and for functional template formation.
Journal: Journal of virology
Published: January 04, 2013
Showing 1-9 of 9
Last Updated: 09/26/2026