Shimin Tao

NHC Key Lab Of Reproduction Regulation
Shanghai, 200237, China

Last Updated: 09/26/2026

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Found 11 publications

Computational Toxicology to Elucidate PFASs Causing Fetal Growth Restriction via Binding to and Degrading IGF1 Protein.

Computational Toxicology to Elucidate PFASs Causing Fetal Growth Restriction via Binding to and Degrading IGF1 Protein.

Journal: Environmental science & technology

Published: November 26, 2025

Nicotinamide improves the impaired extravillous trophoblast cell invasion induced by PM2.5 exposure-associated increase of TNFα secretion through the ROS/NF-κB/FLT1 pathway.

Nicotinamide improves the impaired extravillous trophoblast cell invasion induced by PM2.5 exposure-associated increase of TNFα secretion through the ROS/NF-κB/FLT1 pathway.

Journal: Ecotoxicology and environmental safety

Published: February 07, 2024

Cholinergic anti-inflammatory pathway mediates diesel exhaust PM2.5-induced pulmonary and systemic inflammation.

Cholinergic anti-inflammatory pathway mediates diesel exhaust PM2.5-induced pulmonary and systemic inflammation.

Journal: Journal of hazardous materials

Published: February 21, 2023

Maternal exposure to ambient PM2.5 perturbs the metabolic homeostasis of maternal serum and placenta in mice.

Maternal exposure to ambient PM2.5 perturbs the metabolic homeostasis of maternal serum and placenta in mice.

Journal: Environmental research

Published: July 26, 2022

Maternal exposure to ambient PM2.5 causes fetal growth restriction via the inhibition of spiral artery remodeling in mice.

Maternal exposure to ambient PM2.5 causes fetal growth restriction via the inhibition of spiral artery remodeling in mice.

Journal: Ecotoxicology and environmental safety

Published: November 16, 2021

Concentrated ambient fine particles exposure affects ovarian follicle development in mice.

Concentrated ambient fine particles exposure affects ovarian follicle development in mice.

Journal: Ecotoxicology and environmental safety

Published: October 19, 2021

Exposure to different fractions of diesel exhaust PM2.5 induces different levels of pulmonary inflammation and acute phase response.

Exposure to different fractions of diesel exhaust PM2.5 induces different levels of pulmonary inflammation and acute phase response.

Journal: Ecotoxicology and environmental safety

Published: September 05, 2020

Hypothalamic-pituitary-adrenal axis mediates ambient PM2.5 exposure-induced pulmonary inflammation.

Hypothalamic-pituitary-adrenal axis mediates ambient PM2.5 exposure-induced pulmonary inflammation.

Journal: Ecotoxicology and environmental safety

Published: June 05, 2020

Chronic exposure to diesel exhaust particulate matter impairs meiotic progression during spermatogenesis in a mouse model.

Chronic exposure to diesel exhaust particulate matter impairs meiotic progression during spermatogenesis in a mouse model.

Journal: Ecotoxicology and environmental safety

Published: April 20, 2020

dl-Mandelic acid exhibits high sperm-immobilizing activity and low vaginal irritation: A potential non-surfactant spermicide for contraception.

dl-Mandelic acid exhibits high sperm-immobilizing activity and low vaginal irritation: A potential non-surfactant spermicide for contraception.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: November 07, 2019

Developmental programming of obesity by maternal exposure to concentrated ambient PM2.5 is maternally transmitted into the third generation in a mouse model.

Developmental programming of obesity by maternal exposure to concentrated ambient PM2.5 is maternally transmitted into the third generation in a mouse model.

Journal: Particle and fibre toxicology

Published: December 10, 2018

Showing 1-11 of 11

Last Updated: 09/26/2026