Shingo Arakawa
Shizuoka, Japan
Last Updated: 09/26/2026
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Found 8 publications
Decreased hepatic phosphorylated p38 mitogen-activated protein kinase contributes to attenuation of thioacetamide-induced hepatic necrosis in diet-induced obese mice.
Journal: The Journal of toxicological sciences
Published: March 11, 2016
Utilization of glutathione S-transferase Mu 1- and Theta 1-null mice as animal models for absorption, distribution, metabolism, excretion and toxicity studies.
Journal: Expert opinion on drug metabolism & toxicology
Published: March 20, 2013
Role of connexin 32 in acetaminophen toxicity in a knockout mice model.
Journal: Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie
Published: March 02, 2013
Thioacetamide-induced Hepatocellular Necrosis Is Attenuated in Diet-induced Obese Mice.
Journal: Journal of toxicologic pathology
Published: December 03, 2012
Resistance to acetaminophen-induced hepatotoxicity in glutathione S-transferase Mu 1-null mice.
Journal: The Journal of toxicological sciences
Published: June 13, 2012
Evaluation of hepatic glutathione transferase Mu 1 and Theta 1 activities in humans and mice using genotype information.
Journal: Drug metabolism and disposition: the biological fate of chemicals
Published: December 16, 2011
Methemoglobinemia induced by 1,2-dichloro-4-nitrobenzene in mice with a disrupted glutathione S-transferase Mu 1 gene.
Journal: Drug metabolism and disposition: the biological fate of chemicals
Published: June 22, 2010
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Last Updated: 09/26/2026