Shinya Satoh
Hamamatsu, Japan
Last Updated: 09/26/2026
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Found 37 publications
Assembly of lipid droplet-associated ring structures in hepatitis C virus-infected cells via liquid-liquid phase separation (LLPS) and non-LLPS mechanisms.
Journal: Journal of virology
Published: July 01, 2026
Identification of an isoquinoline-derived quinazoline hybrid as a transcriptional inhibitor of fatty acid synthase to suppress hepatitis C virus production.
Journal: Biochemical and biophysical research communications
Published: March 16, 2026
FGF21 upregulation by hepatitis C virus via the eIF2α-ATF4 pathway: implications for interferon signaling suppression and TRIM31-mediated TSC degradation.
Journal: Frontiers in microbiology
Published: June 28, 2024
Involvement of ribosomal protein L17 and Y-box binding protein 1 in the assembly of hepatitis C virus potentially via their interaction with the 3' untranslated region of the viral genome.
Journal: Journal of virology
Published: June 20, 2024
Antiviral mechanism of preclinical antimalarial compounds possessing multiple antiviral activities.
Journal: FASEB bioAdvances
Published: October 27, 2020
Identification of ribavirin-responsive cis-elements for GPAM suppression in the GPAM genome.
Journal: Biochemical and biophysical research communications
Published: August 21, 2020
Recurrence, death, and secondary malignancy after ovarian conservation for young women with early-stage low-grade endometrial cancer.
Journal: Gynecologic oncology
Published: June 09, 2019
A new hepatoma cell line exhibiting high susceptibility to hepatitis B virus infection.
Journal: Biochemical and biophysical research communications
Published: May 14, 2019
Tumor characteristics and outcome of uterine carcinosarcoma in women aged ≥80 years.
Journal: Surgical oncology
Published: September 25, 2018
High-level expression of STING restricts susceptibility to HBV by mediating type III IFN induction.
Journal: FASEB bioAdvances
Published: September 21, 2018
Daunorubicin, a topoisomerase II poison, suppresses viral production of hepatitis B virus by inducing cGAS-dependent innate immune response.
Journal: Biochemical and biophysical research communications
Published: August 24, 2018
Last Updated: 09/26/2026