Shulamit B. Dayan-Wallach

Jerusalem, JM, Israel

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 20 publications

Targeting the endocannabinoid system to suppress mTORC1 hyperactivation in TSC-associated kidney disease.

Targeting the endocannabinoid system to suppress mTORC1 hyperactivation in TSC-associated kidney disease.

Journal: American journal of physiology. Renal physiology

Published: July 24, 2025

Therapeutic potential of cannabinoid receptor 2 agonist osteogenic growth peptide in lung fibrosis in aged mice.

Therapeutic potential of cannabinoid receptor 2 agonist osteogenic growth peptide in lung fibrosis in aged mice.

Journal: American journal of respiratory cell and molecular biology

Published: March 04, 2025

MTADV 5-MER peptide suppresses chronic inflammations as well as autoimmune pathologies and unveils a new potential target-Serum Amyloid A.

MTADV 5-MER peptide suppresses chronic inflammations as well as autoimmune pathologies and unveils a new potential target-Serum Amyloid A.

Journal: Journal of autoimmunity

Published: June 09, 2021

sFasL-The Key to a Riddle: Immune Responses in Aging Lung and Disease.

sFasL-The Key to a Riddle: Immune Responses in Aging Lung and Disease.

Journal: International journal of molecular sciences

Published: December 06, 2020

CMH-Small Molecule Docks into SIRT1, Elicits Human IPF-Lung Fibroblast Cell Death, Inhibits Ku70-deacetylation, FLIP and Experimental Pulmonary Fibrosis.

CMH-Small Molecule Docks into SIRT1, Elicits Human IPF-Lung Fibroblast Cell Death, Inhibits Ku70-deacetylation, FLIP and Experimental Pulmonary Fibrosis.

Journal: Biomolecules

Published: May 07, 2020

SIRT1 Deficiency, Specifically in Fibroblasts, Decreases Apoptosis Resistance and Is Associated with Resolution of Lung-Fibrosis.

SIRT1 Deficiency, Specifically in Fibroblasts, Decreases Apoptosis Resistance and Is Associated with Resolution of Lung-Fibrosis.

Journal: Biomolecules

Published: May 07, 2020

Forefront: MiR-34a-Knockout Mice with Wild Type Hematopoietic Cells, Retain Persistent Fibrosis Following Lung Injury.

Forefront: MiR-34a-Knockout Mice with Wild Type Hematopoietic Cells, Retain Persistent Fibrosis Following Lung Injury.

Journal: International journal of molecular sciences

Published: February 23, 2020

Senescence, the Janus of Lung Injury and Repair.

Senescence, the Janus of Lung Injury and Repair.

Journal: American journal of respiratory cell and molecular biology

Published: January 25, 2020

Matrix Metalloproteinases Retain Soluble FasL-mediated Resistance to Cell Death in Fibrotic-Lung Myofibroblasts.

Matrix Metalloproteinases Retain Soluble FasL-mediated Resistance to Cell Death in Fibrotic-Lung Myofibroblasts.

Journal: Cells

Published: January 16, 2020

Increased Regeneration Following Stress-Induced Lung Injury in Bleomycin-Treated Chimeric Mice with CD44 Knockout Mesenchymal Cells.

Increased Regeneration Following Stress-Induced Lung Injury in Bleomycin-Treated Chimeric Mice with CD44 Knockout Mesenchymal Cells.

Journal: Cells

Published: September 08, 2019

The Role of Telomerase and Telomeres in Interstitial Lung Diseases: From Molecules to Clinical Implications.

The Role of Telomerase and Telomeres in Interstitial Lung Diseases: From Molecules to Clinical Implications.

Journal: International journal of molecular sciences

Published: May 23, 2019

A Profibrotic Phenotype in Naïve and in Fibrotic Lung Myofibroblasts Is Governed by Modulations in Thy-1 Expression and Activation.

A Profibrotic Phenotype in Naïve and in Fibrotic Lung Myofibroblasts Is Governed by Modulations in Thy-1 Expression and Activation.

Journal: Mediators of inflammation

Published: December 18, 2017

Showing 1-12 of 20

Last Updated: 09/26/2026