Shunichi Tayama

Sendai, 9808575, Japan

Last Updated: 09/26/2026

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Found 11 publications

Circulating memory-phenotype CD4+ T cells rapidly accumulate in the ischemic gut to exacerbate tissue injury in an innate manner.

Circulating memory-phenotype CD4+ T cells rapidly accumulate in the ischemic gut to exacerbate tissue injury in an innate manner.

Journal: Cell reports
Published: August 11, 2025

Naturally arising memory-phenotype CD4+ T lymphocytes contain an undifferentiated population that can generate TH1, TH17, and Treg cells.

Naturally arising memory-phenotype CD4+ T lymphocytes contain an undifferentiated population that can generate TH1, TH17, and Treg cells.

Journal: Science advances
Published: December 04, 2024

Supersulfide controls intestinal inflammation by suppressing CD4+ T cell proliferation.

Supersulfide controls intestinal inflammation by suppressing CD4+ T cell proliferation.

Journal: Frontiers in immunology
Published: October 05, 2024

Naturally Arising Memory-Phenotype CD4+ T Lymphocytes Give Rise to Multiple Helper Subsets to Contribute to Tumor Immunity while Inhibiting GVHD.

Naturally Arising Memory-Phenotype CD4+ T Lymphocytes Give Rise to Multiple Helper Subsets to Contribute to Tumor Immunity while Inhibiting GVHD.

Journal: Cancer immunology research
Published: June 27, 2024

Excess generation and activation of naturally arising memory-phenotype CD4+ T lymphocytes are inhibited by regulatory T cells in steady state.

Excess generation and activation of naturally arising memory-phenotype CD4+ T lymphocytes are inhibited by regulatory T cells in steady state.

Journal: Frontiers in immunology
Published: May 09, 2024

Redefining the Foreign Antigen and Self-Driven Memory CD4+ T-Cell Compartments via Transcriptomic, Phenotypic, and Functional Analyses.

Redefining the Foreign Antigen and Self-Driven Memory CD4+ T-Cell Compartments via Transcriptomic, Phenotypic, and Functional Analyses.

Journal: Frontiers in immunology
Published: February 07, 2022

Fatty acid-binding protein 5 limits ILC2-mediated allergic lung inflammation in a murine asthma model.

Fatty acid-binding protein 5 limits ILC2-mediated allergic lung inflammation in a murine asthma model.

Journal: Scientific reports
Published: June 06, 2020

Correction: TRAF5 Deficiency Ameliorates the Severity of Dextran Sulfate Sodium Colitis by Decreasing TRAF2 Expression in Nonhematopoietic Cells.

Correction: TRAF5 Deficiency Ameliorates the Severity of Dextran Sulfate Sodium Colitis by Decreasing TRAF2 Expression in Nonhematopoietic Cells.

Journal: ImmunoHorizons
Published: April 25, 2020

Fatty acid-binding protein 3 regulates differentiation of IgM-producing plasma cells.

Fatty acid-binding protein 3 regulates differentiation of IgM-producing plasma cells.

Journal: The FEBS journal
Published: April 08, 2020

TRAF5 Deficiency Ameliorates the Severity of Dextran Sulfate Sodium Colitis by Decreasing TRAF2 Expression in Nonhematopoietic Cells.

TRAF5 Deficiency Ameliorates the Severity of Dextran Sulfate Sodium Colitis by Decreasing TRAF2 Expression in Nonhematopoietic Cells.

Journal: ImmunoHorizons
Published: January 24, 2020

IQ motif-containing GTPase-activating protein 1 is essential for the optimal maintenance of lung ILC2s.

IQ motif-containing GTPase-activating protein 1 is essential for the optimal maintenance of lung ILC2s.

Journal: International immunology
Published: October 03, 2019
Showing 1-11 of 11

Last Updated: 09/26/2026