Shunichi Yokoe

Osaka, Japan

Last Updated: 09/26/2026

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Found 16 publications

Corrigendum to "Rapamycin and Starvation Mitigate Indomethacin-Induced Intestinal Damage through Preservation of Lysosomal Vacuolar ATPase Integrity": [The Journal of Pharmacology and Experimental Therapeutics 390 (2024) 108-115].

Corrigendum to "Rapamycin and Starvation Mitigate Indomethacin-Induced Intestinal Damage through Preservation of Lysosomal Vacuolar ATPase Integrity": [The Journal of Pharmacology and Experimental Therapeutics 390 (2024) 108-115].

Journal: The Journal of pharmacology and experimental therapeutics

Published: August 30, 2025

Beneficial Effects of Chymase Inhibition on Cardiac Diastolic Function and Remodeling Induced by Chronic Angiotensin II Stimulation.

Beneficial Effects of Chymase Inhibition on Cardiac Diastolic Function and Remodeling Induced by Chronic Angiotensin II Stimulation.

Journal: International journal of molecular sciences

Published: July 25, 2025

TMEM182 inhibits myocardial differentiation of human iPS cells by maintaining the activated state of Wnt/β-catenin signaling through an increase in ILK expression.

TMEM182 inhibits myocardial differentiation of human iPS cells by maintaining the activated state of Wnt/β-catenin signaling through an increase in ILK expression.

Journal: FASEB bioAdvances

Published: June 06, 2024

Rapamycin and Starvation Mitigate Indomethacin-Induced Intestinal Damage through Preservation of Lysosomal Vacuolar ATPase Integrity.

Rapamycin and Starvation Mitigate Indomethacin-Induced Intestinal Damage through Preservation of Lysosomal Vacuolar ATPase Integrity.

Journal: The Journal of pharmacology and experimental therapeutics

Published: October 24, 2023

O-GlcNAcylation-induced GSK-3β activation deteriorates pressure overload-induced heart failure via lack of compensatory cardiac hypertrophy in mice.

O-GlcNAcylation-induced GSK-3β activation deteriorates pressure overload-induced heart failure via lack of compensatory cardiac hypertrophy in mice.

Journal: Frontiers in endocrinology

Published: December 12, 2022

Augmented O-GlcNAcylation exacerbates right ventricular dysfunction and remodeling via enhancement of hypertrophy, mitophagy, and fibrosis in mice exposed to long-term intermittent hypoxia.

Augmented O-GlcNAcylation exacerbates right ventricular dysfunction and remodeling via enhancement of hypertrophy, mitophagy, and fibrosis in mice exposed to long-term intermittent hypoxia.

Journal: Hypertension research : official journal of the Japanese Society of Hypertension

Published: April 20, 2022

Fluctuation in O-GlcNAcylation inactivates STIM1 to reduce store-operated calcium ion entry via down-regulation of Ser621 phosphorylation.

Fluctuation in O-GlcNAcylation inactivates STIM1 to reduce store-operated calcium ion entry via down-regulation of Ser621 phosphorylation.

Journal: The Journal of biological chemistry

Published: May 08, 2020

Overexpression of Na+/H+ exchanger 1 specifically induces cell death in human iPS cells via sustained activation of the Rho kinase ROCK.

Overexpression of Na+/H+ exchanger 1 specifically induces cell death in human iPS cells via sustained activation of the Rho kinase ROCK.

Journal: The Journal of biological chemistry

Published: July 23, 2019

Augmented O-GlcNAcylation attenuates intermittent hypoxia-induced cardiac remodeling through the suppression of NFAT and NF-κB activities in mice.

Augmented O-GlcNAcylation attenuates intermittent hypoxia-induced cardiac remodeling through the suppression of NFAT and NF-κB activities in mice.

Journal: Hypertension research : official journal of the Japanese Society of Hypertension

Published: March 01, 2019

Interleukin-18 in cardiomyocyte: A novel therapeutic target for attenuating cardiac remodeling. Response.

Interleukin-18 in cardiomyocyte: A novel therapeutic target for attenuating cardiac remodeling. Response.

Journal: International journal of cardiology

Published: November 20, 2017

Phospholamban Is Downregulated by pVHL-Mediated Degradation through Oxidative Stress in Failing Heart.

Phospholamban Is Downregulated by pVHL-Mediated Degradation through Oxidative Stress in Failing Heart.

Journal: International journal of molecular sciences

Published: September 14, 2017

Interleukin-18 gene deletion protects against sepsis-induced cardiac dysfunction by inhibiting PP2A activity.

Interleukin-18 gene deletion protects against sepsis-induced cardiac dysfunction by inhibiting PP2A activity.

Journal: International journal of cardiology

Published: October 07, 2016

Showing 1-12 of 16

Last Updated: 09/26/2026