Silvia Scaricamazza

Rome, 00133, Italy

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 27 publications

Transcriptomic Analysis Reveals the Beneficial Effects of Spermidine in an ALS Mouse Model.

Transcriptomic Analysis Reveals the Beneficial Effects of Spermidine in an ALS Mouse Model.

Journal: Biomolecules

Published: March 11, 2026

Transcriptomic Analysis Reveals the Beneficial Effects of Spermidine in an ALS Mouse Model.

Transcriptomic Analysis Reveals the Beneficial Effects of Spermidine in an ALS Mouse Model.

Journal: Biomolecules

Published: March 11, 2026

The hypothalamus is an early site of mitochondrial failure and neuro-immune circuit disruption in amyotrophic lateral sclerosis.

The hypothalamus is an early site of mitochondrial failure and neuro-immune circuit disruption in amyotrophic lateral sclerosis.

Journal: Molecular metabolism

Published: January 20, 2026

Effects of neuromelanin buildup in rodent nigral dopamine neurons: implications for sex-biased vulnerability in Parkinson's disease.

Effects of neuromelanin buildup in rodent nigral dopamine neurons: implications for sex-biased vulnerability in Parkinson's disease.

Journal: Neurobiology of disease

Published: December 17, 2025

Brown Adipose Tissue undergoes pathological perturbations and shapes C2C12 myoblast homeostasis in the SOD1-G93A mouse model of Amyotrophic Lateral Sclerosis.

Brown Adipose Tissue undergoes pathological perturbations and shapes C2C12 myoblast homeostasis in the SOD1-G93A mouse model of Amyotrophic Lateral Sclerosis.

Journal: Heliyon

Published: October 02, 2024

Hidden players in the metabolic vulnerabilities of amyotrophic lateral sclerosis.

Hidden players in the metabolic vulnerabilities of amyotrophic lateral sclerosis.

Journal: Trends in endocrinology and metabolism: TEM

Published: October 02, 2024

Polyamine metabolism dysregulation contributes to muscle fiber vulnerability in ALS.

Polyamine metabolism dysregulation contributes to muscle fiber vulnerability in ALS.

Journal: Cell reports

Published: July 04, 2024

Physical exercise in amyotrophic lateral sclerosis: a potential co-adjuvant therapeutic option to counteract disease progression.

Physical exercise in amyotrophic lateral sclerosis: a potential co-adjuvant therapeutic option to counteract disease progression.

Journal: Frontiers in cell and developmental biology

Published: April 22, 2024

N-acetylaspartate promotes glycolytic-to-oxidative fiber-type switch and resistance to atrophic stimuli in myotubes.

N-acetylaspartate promotes glycolytic-to-oxidative fiber-type switch and resistance to atrophic stimuli in myotubes.

Journal: Cell death & disease

Published: April 09, 2024

Trimetazidine Improves Mitochondrial Dysfunction in SOD1G93A Cellular Models of Amyotrophic Lateral Sclerosis through Autophagy Activation.

Trimetazidine Improves Mitochondrial Dysfunction in SOD1G93A Cellular Models of Amyotrophic Lateral Sclerosis through Autophagy Activation.

Journal: International journal of molecular sciences

Published: January 24, 2024

Endurance exercise has a negative impact on the onset of SOD1-G93A ALS in female mice and affects the entire skeletal muscle-motor neuron axis.

Endurance exercise has a negative impact on the onset of SOD1-G93A ALS in female mice and affects the entire skeletal muscle-motor neuron axis.

Journal: Frontiers in pharmacology

Published: December 22, 2023

Correction: Immune-mediated myogenesis and acetylcholine receptor clustering promote a slow disease progression in ALS mouse models.

Correction: Immune-mediated myogenesis and acetylcholine receptor clustering promote a slow disease progression in ALS mouse models.

Journal: Inflammation and regeneration

Published: April 19, 2023

Showing 1-12 of 27

Last Updated: 09/26/2026