Simon Lammle

Dresden, SN, Germany

Last Updated: 09/26/2026

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Found 8 publications

CNP Promotes Antiarrhythmic Effects via Phosphodiesterase 2.

CNP Promotes Antiarrhythmic Effects via Phosphodiesterase 2.

Journal: Circulation research

Published: January 30, 2023

The reduced activity of PP-1α under redox stress condition is a consequence of GSH-mediated transient disulfide formation.

The reduced activity of PP-1α under redox stress condition is a consequence of GSH-mediated transient disulfide formation.

Journal: Scientific reports

Published: April 04, 2018

Erratum to: Differential regulation of protein phosphatase 1 (PP1) isoforms in human heart failure and atrial fibrillation.

Erratum to: Differential regulation of protein phosphatase 1 (PP1) isoforms in human heart failure and atrial fibrillation.

Journal: Basic research in cardiology

Published: July 09, 2017

Differential regulation of protein phosphatase 1 (PP1) isoforms in human heart failure and atrial fibrillation.

Differential regulation of protein phosphatase 1 (PP1) isoforms in human heart failure and atrial fibrillation.

Journal: Basic research in cardiology

Published: April 29, 2017

Phosphodiesterase 2 Protects Against Catecholamine-Induced Arrhythmia and Preserves Contractile Function After Myocardial Infarction.

Phosphodiesterase 2 Protects Against Catecholamine-Induced Arrhythmia and Preserves Contractile Function After Myocardial Infarction.

Journal: Circulation research

Published: October 02, 2016

Calcium/Calmodulin-Dependent Protein Kinase II Activity Persists During Chronic β-Adrenoceptor Blockade in Experimental and Human Heart Failure.

Calcium/Calmodulin-Dependent Protein Kinase II Activity Persists During Chronic β-Adrenoceptor Blockade in Experimental and Human Heart Failure.

Journal: Circulation. Heart failure

Published: May 05, 2016

Physiologic force-frequency response in engineered heart muscle by electromechanical stimulation.

Physiologic force-frequency response in engineered heart muscle by electromechanical stimulation.

Journal: Biomaterials

Published: January 02, 2015

PDE2-mediated cAMP hydrolysis accelerates cardiac fibroblast to myofibroblast conversion and is antagonized by exogenous activation of cGMP signaling pathways.

PDE2-mediated cAMP hydrolysis accelerates cardiac fibroblast to myofibroblast conversion and is antagonized by exogenous activation of cGMP signaling pathways.

Journal: American journal of physiology. Heart and circulatory physiology

Published: February 18, 2014

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Last Updated: 09/26/2026