Sirui Liu

52 Meihua Dong Road
Zhuhai, 519000, China

Last Updated: 09/26/2026

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Found 11 publications

Abnormal circadian pattern of urinary sodium excretion and chronic kidney disease progression.

Abnormal circadian pattern of urinary sodium excretion and chronic kidney disease progression.

Journal: BMC nephrology

Published: September 03, 2025

SGLT2 inhibition attenuates diabetic tubulopathy by suppressing SGK1-mediated pyroptosis.

SGLT2 inhibition attenuates diabetic tubulopathy by suppressing SGK1-mediated pyroptosis.

Journal: Frontiers in endocrinology

Published: April 29, 2025

Inhibition of SGLT2 protects podocytes in diabetic kidney disease by rebalancing mitochondria-associated endoplasmic reticulum membranes.

Inhibition of SGLT2 protects podocytes in diabetic kidney disease by rebalancing mitochondria-associated endoplasmic reticulum membranes.

Journal: Cell communication and signaling : CCS

Published: July 02, 2024

ER Stress-Perturbed Intracellular Protein O-GlcNAcylation Aggravates Podocyte Injury in Diabetes Nephropathy.

ER Stress-Perturbed Intracellular Protein O-GlcNAcylation Aggravates Podocyte Injury in Diabetes Nephropathy.

Journal: International journal of molecular sciences

Published: October 30, 2023

(Pro)renin receptor mediates tubular epithelial cell pyroptosis in diabetic kidney disease via DPP4-JNK pathway.

(Pro)renin receptor mediates tubular epithelial cell pyroptosis in diabetic kidney disease via DPP4-JNK pathway.

Journal: Journal of translational medicine

Published: October 02, 2023

β-catenin-inhibited Sumoylation modification of LKB1 and fatty acid metabolism is critical in renal fibrosis.

β-catenin-inhibited Sumoylation modification of LKB1 and fatty acid metabolism is critical in renal fibrosis.

Journal: Cell death & disease

Published: September 15, 2023

24-h central pressure is a valuable predictor for left ventricular hypertrophy in non-dialysis patients with chronic kidney disease.

24-h central pressure is a valuable predictor for left ventricular hypertrophy in non-dialysis patients with chronic kidney disease.

Journal: Hypertension research : official journal of the Japanese Society of Hypertension

Published: September 01, 2023

Site 1 protease aggravates acute kidney injury by promoting tubular epithelial cell ferroptosis through SIRT3-SOD2-mtROS signaling.

Site 1 protease aggravates acute kidney injury by promoting tubular epithelial cell ferroptosis through SIRT3-SOD2-mtROS signaling.

Journal: The FEBS journal

Published: May 30, 2023

Mitochondria-associated endoplasmic reticulum membranes promote mitochondrial fission through AKAP1-Drp1 pathway in podocytes under high glucose conditions.

Mitochondria-associated endoplasmic reticulum membranes promote mitochondrial fission through AKAP1-Drp1 pathway in podocytes under high glucose conditions.

Journal: Experimental cell research

Published: November 22, 2022

Increased thromboxane/prostaglandin receptors contribute to high glucose-induced podocyte injury and mitochondrial fission through ROCK1-Drp1 signaling.

Increased thromboxane/prostaglandin receptors contribute to high glucose-induced podocyte injury and mitochondrial fission through ROCK1-Drp1 signaling.

Journal: The international journal of biochemistry & cell biology

Published: March 16, 2022

Increased long noncoding RNA maternally expressed gene 3 contributes to podocyte injury induced by high glucose through regulation of mitochondrial fission.

Increased long noncoding RNA maternally expressed gene 3 contributes to podocyte injury induced by high glucose through regulation of mitochondrial fission.

Journal: Cell death & disease

Published: May 28, 2020

Showing 1-11 of 11

Last Updated: 09/26/2026