So J. Park

Cambridge, ENG, United Kingdom

Last Updated: 09/26/2026

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Found 21 publications

Author Correction: p300 nucleocytoplasmic shuttling underlies mTORC1 hyperactivation in Hutchinson-Gilford progeria syndrome.

Author Correction: p300 nucleocytoplasmic shuttling underlies mTORC1 hyperactivation in Hutchinson-Gilford progeria syndrome.

Journal: Nature cell biology
Published: November 29, 2025

How does autophagy impact neurological function?

How does autophagy impact neurological function?

Journal: The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry
Published: March 13, 2025

Alpha-synuclein mutations mislocalize cytoplasmic p300 compromising autophagy, which is rescued by ACLY inhibition.

Alpha-synuclein mutations mislocalize cytoplasmic p300 compromising autophagy, which is rescued by ACLY inhibition.

Journal: Neuron
Published: October 22, 2024

Transferrin receptor controls both autophagosome formation and closure via phosphatidylinositol 3-phosphate synthesis.

Transferrin receptor controls both autophagosome formation and closure via phosphatidylinositol 3-phosphate synthesis.

Journal: Developmental cell
Published: August 07, 2024

Nuclear proteasomes buffer cytoplasmic proteins during autophagy compromise.

Nuclear proteasomes buffer cytoplasmic proteins during autophagy compromise.

Journal: Nature cell biology
Published: June 21, 2023

p300 nucleocytoplasmic shuttling underlies mTORC1 hyperactivation in Hutchinson-Gilford progeria syndrome.

p300 nucleocytoplasmic shuttling underlies mTORC1 hyperactivation in Hutchinson-Gilford progeria syndrome.

Journal: Nature cell biology
Published: May 12, 2023

Increased SORBS3 expression in brain ageing contributes to autophagic decline via YAP1-WWTR1/TAZ signaling.

Increased SORBS3 expression in brain ageing contributes to autophagic decline via YAP1-WWTR1/TAZ signaling.

Journal: Autophagy
Published: July 13, 2022

The complexity of biological control systems: An autophagy case study.

The complexity of biological control systems: An autophagy case study.

Journal: BioEssays : news and reviews in molecular, cellular and developmental biology
Published: September 21, 2021

Cell type-specific YAP1-WWTR1/TAZ transcriptional responses after autophagy perturbations are determined by levels of α-catenins (CTNNA1 and CTNNA3).

Cell type-specific YAP1-WWTR1/TAZ transcriptional responses after autophagy perturbations are determined by levels of α-catenins (CTNNA1 and CTNNA3).

Journal: Autophagy
Published: May 26, 2021

Vinexin contributes to autophagic decline in brain ageing across species.

Vinexin contributes to autophagic decline in brain ageing across species.

Journal: Cell death and differentiation
Published: April 23, 2021

Depletion of HNRNPA1 induces peroxisomal autophagy by regulating PEX1 expression.

Depletion of HNRNPA1 induces peroxisomal autophagy by regulating PEX1 expression.

Journal: Biochemical and biophysical research communications
Published: January 22, 2021

Increased O-GlcNAcylation of Drp1 by amyloid-beta promotes mitochondrial fission and dysfunction in neuronal cells.

Increased O-GlcNAcylation of Drp1 by amyloid-beta promotes mitochondrial fission and dysfunction in neuronal cells.

Journal: Molecular brain
Published: December 14, 2020
Showing 1-12 of 21

Last Updated: 09/26/2026